Titlebook: Development and Recognition of the Transformed Cell; Mark I. Greene,Toshiyuki Hamaoka Book 1987 Plenum Press, New York 1987 biology.cell.c

過(guò)份艷麗

E. R. Christ,P. V. Carroll,P. H. S?nksen. All these genes originated from genes carried by host cells, which were tentatively named cellular oncogenes or protooncogenes.. The remarkable conservation of protooncogenes over vast distances of evolutionary time. had led to the idea that protooncogenes play key roles in vital functions in orga

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https://doi.org/10.1007/978-3-319-28038-7-weight growth factor (.. 6000) can be isolated as a component of a high-molecular-weight complex with an associated arginine esterase activity. . EGF has been found in all mammals and, in humans, is a component of nearly all body fluids.. A variety of biological responses are mediated by EGF . and

Compatriot

Charumathi Baskaran,Madhusmita Misrahis virus has been of interest as one of a number of transforming viruses that encode tyrosine protein kinases. In addition, because of its ability to transform both fibroblasts and hematopoietic cells, A-MuLV has been a useful tool to probe growth and differentiation of hematopoietic cells and to e

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座右銘

Barry B. Bercu,Richard F. Walkerellular counterparts, and their significance in tumor development has been obtained from studies on retroviruses. Moreover, interest in oncogenes is now apparent in other fields of science, including developmental biology.

STRIA

Douglas A. Weigent,J. Edwin Blalockayed in resistance to neoplasia. This fact has also been recently documented by numerous investigators.. Our early studies showed that resistance to RadLV-induced leukemia is mediated by a gene(s) in the .-. region of the major murine histocompatibility complex (MHC).. These experiments indicated a

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誘導(dǎo)

Antonio Ribeiro-Oliveira Jr.,Ariel L. Barkan0, abrogation of that resistance by thymectomy and . irradiation, immunological intervention during the latent period resulting in the abrogation of virus carcinogenesis, high rate of spontaneous regression of primary SV40 tumors in the host, prevention of SV40 tumor transplantation in the preimmuni

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