Titlebook: Dementia in Parkinsonism; S. E. Daniel,F. F. Cruz-Sánchez,A. J. Lees Conference proceedings 1997 Springer-Verlag Wien 1997 Alzheimer.Lipop

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collagen

Barbara Brandstetter,Steffen Rangee of Lewy bodies, as other areas of the brain containing Lewy bodies do not show evidence of either oxidative stress or mitochondrial dysfunction. Oxidative stress has now been demonstrated in Alzheimer’s disease and its presence appears to be correlated with regions of marked pathological changes.

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,Indices of oxidative stress in Parkinson’s disease, Alzheimer’s disease and dementia with Lewy bodie of Lewy bodies, as other areas of the brain containing Lewy bodies do not show evidence of either oxidative stress or mitochondrial dysfunction. Oxidative stress has now been demonstrated in Alzheimer’s disease and its presence appears to be correlated with regions of marked pathological changes.

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,Apolipoprotein E gene in Parkinson’s disease, Lewy body dementia and Alzheimer’s disease, (n = 175) compared to aged non-demented controls (n = 47) and the Scottish population (n = 400). We conclude that Lewy body disease should be regarded as a variant of Alzheimer’s but not Parkinson’s disease.

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https://doi.org/10.1007/978-3-322-84159-9ation with dementia in Parkinsonism. Plaques can be associated with cortical Lewy bodies and, the contribution of each to dementing processes remains unresolved. In a consecutive autopsy series of 610 patients with parkinsonism, the total prevalence of retrospectively assessed dementia was 34.6%. In

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