Titlebook: Delayed Preconditioning and Adaptive Cardioprotection; G. F. Baxter,D. M. Yellon Book 1998 Springer Science+Business Media Dordrecht 1998

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https://doi.org/10.1007/978-3-322-99076-1hours after the initial nonlethal ischaemia. This late effect of ischaemic preconditioning was named the second window of protection [16]. Although de novo protein synthesis is not involved in the mechanism of classic preconditioning [17], the time course of the second window suggests that the synth

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https://doi.org/10.1007/978-3-322-99003-7 it is formed; the duration of its effects are very transient with a half-life in human blood of less than one second [3]. Thus adenosine is considered a short-term mediator that maintains the metabolic equilibrium of the heart within a time frame of seconds to minutes.

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https://doi.org/10.1007/978-3-031-28844-9ossibility that angina heralding MI may represent the clinical correlate of ischaemic preconditioning is extremely appealing because of its inherent cardioprotective action. Therefore, this chapter will review the concept that angina pectoris before MI may exert a protective role, through an adaptiv

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Delayed Preconditioning Against Lethal Ischaemic Injury, 7), endotoxin and its derivatives (chapter 9), and adenosine analogues (chapter 10). In this chapter, we will review the background to the discovery of delayed preconditioning, the characteristics of this phenomenon and possible mechanisms. We will concentrate particularly on the direct cytoprotect

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Changes in Cardiac Gene Expression After Ischaemia and Reperfusion, or surgery, and to understand the adaptive processes that may protect myocardial function. Few studies of gene transcription have, however, been conducted using human tissue, due to the limitations of working with living subjects. As a result, our understanding of the transcriptional response of my

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, Role of Manganese Superoxide Dismutase in Delayed Preconditioning,hours after the initial nonlethal ischaemia. This late effect of ischaemic preconditioning was named the second window of protection [16]. Although de novo protein synthesis is not involved in the mechanism of classic preconditioning [17], the time course of the second window suggests that the synth

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