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Titlebook: Degenerative Diseases of the Retina; Robert E. Anderson,Matthew M. LaVail,Joe G. Hollyf Book 1995 Springer Science+Business Media New York

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發(fā)表于 2025-3-21 19:38:53 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Degenerative Diseases of the Retina
編輯Robert E. Anderson,Matthew M. LaVail,Joe G. Hollyf
視頻videohttp://file.papertrans.cn/265/264869/264869.mp4
圖書封面Titlebook: Degenerative Diseases of the Retina;  Robert E. Anderson,Matthew M. LaVail,Joe G. Hollyf Book 1995 Springer Science+Business Media New York
描述In 1984, we organized a two-day symposium on retinal degenerations as part of the biennial meeting of the VI International Society for Eye Research, held in Alicante, Spain. The success of this first meeting led to the second held, two years later in Sendai, Japan, organized as a satellite of the VII ISER. We were fortunate that these meetings began at a time of vigorous research activity in the area of retinal degenerations, thanks to the financial support of the Retinitis Pigmentosa Foundation and the strong encouragement of its scientific director, Dr. Alan Laties. Significant advances were made so that every two years scientists were eager to meet to share their findings. The programs included presentations by both basic and clinical researchers with ample time for informal discussions in a relaxed atmos- phere. Many investigators met for the first time at these symposia and a number of fruitful collaborations were established. This book contains the proceedings of the VI International Symposium on Retinal Degenerations held November 6-10, 1994, in Jerusalem. As with the other meetings, some new areas were covered. One session was devoted to apoptosis, an important process invo
出版日期Book 1995
關鍵詞clinical research; cytokine; cytokines; death; eye; growth; lead; mutation; phenotype; receptor; research; reti
版次1
doihttps://doi.org/10.1007/978-1-4615-1897-6
isbn_softcover978-1-4613-5774-2
isbn_ebook978-1-4615-1897-6
copyrightSpringer Science+Business Media New York 1995
The information of publication is updating

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https://doi.org/10.1007/978-3-662-67693-6lux and 3000 lux diffuse, white fluorescent light for 2 h). At higher illuminances and extended post — exposure intervals, necrotic cell death is prominent. In our model, the threshold for apoptosis is at 1000 lux for 2 hrs. Distinct morphological signs of apoptosis with chromatin — and cytoplasmic
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https://doi.org/10.1007/978-3-322-96986-6d photoreceptor degeneration remains unknown, certain hypotheses were made based on previous animal studies [1-8]. Free radical formation and lipid peroxidation are among the most widely accepted hypotheses regarding the pathogenesis of photic retinopathy [1-5]. In addition, possible roles for prote
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https://doi.org/10.1007/978-3-322-96986-6 cell death of photoreceptors by apoptosis. The interstitial retinol binding protein (IRBP) promoter was used to drive expression of the human papilloma virus 16 (HPV 16) E7 gene in the retina and other ocular tissues in mice. The result is the death of photoreceptors as they undergo terminal differ
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Die Herrschaft der Reflexionseliteit plays a critical role in retinal homeostasis. RPE cells are multifunctional in nature and have been compared to machropages (Elner et al., 1981, Young and Bok, 1969), oligodendrocytes (Steinberg and Wood, 1974), astrocytes (Immel and Steinberg, 1986), melanocytes (Feeny-Burns, 1980) and hepatocyt
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Methodische Grunds?tze der Arbeitsbewertungs. Over the past 5 years, significant progress has been made elucidating the mechanism of NO synthesis and the functions of NO in different biological systems. NO is produced by cells, and serves a wide variety of functions in different cells, ranging from vascular endothelia, immune cells, neurons
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Begriffliche und thematische Abgrenzungen and recessive traits were similar, mapping of genes proceeded at similar rates, and comparative mapping produced surprises through the discovery of the large number and size of mouse and human homologous chromosomal segments retained since the separation of the species 65 million years ago. What wa
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Die Arbeitswelt im 21. Jahrhundertmembrane (BM), were studied in rat and rabbit. Disintegration of the regular arrangement of RPE basal infoldings, a massive increase in basal lamina-like deposits, and the presence of unusual collagen polymers are common features in both rodent species. In the rat, minor strain-specific variations e
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