Titlebook: Death Receptors in Cancer Therapy; Wafik S. El-Deiry Book 2005 Humana Press 2005 TNF.apoptosis.carcinoma.cell.cell death.gene therapy.inte

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書目名稱Death Receptors in Cancer Therapy影響因子(影響力)




書目名稱Death Receptors in Cancer Therapy影響因子(影響力)學(xué)科排名




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書目名稱Death Receptors in Cancer Therapy網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Death Receptors in Cancer Therapy被引頻次




書目名稱Death Receptors in Cancer Therapy被引頻次學(xué)科排名




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書目名稱Death Receptors in Cancer Therapy年度引用學(xué)科排名




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書目名稱Death Receptors in Cancer Therapy讀者反饋學(xué)科排名

Hot-Flash

https://doi.org/10.1007/978-3-030-22094-5 None of the other members of the TNFR family identified to date contain recognizable intracellular domains, and instead present binding sites for interactions with TNF receptor-associated factors (TRAFs) and other adaptor proteins.

Ointment

FAR

Mammalian Cell Death Pathways,immune surveillance/suppression of cancer. Caspase activation is highly regulated and defects at virtually all levels of death regulation are observed in cancer. This chapter focuses on the cell biology, biochemistry, and genetics of programmed cell death.

CLAY

Death Signaling and Therapeutic Applications of TRAIL,ecules, ligand-type cytokine molecules including the tumor necrosis factor (TNF) family members have been best characterized. The TNF family members most extensively characterized for death signaling and structure include TNF-α, Fas ligand (FasL), and TNF-related apoptosis-inducing ligand (TRAIL).

朋黨派系

朋黨派系

Regulation of Death Receptors by Synthetic Retinoids,s such as acne and psoriasis, and in the prevention or treatment of certain types of cancer, such as the treatment of acute promyelocytic leukemia (APL) and cutaneous T-cell lymphoma, reversal of premalignant lesions, and inhibition of the development of second primary tumors (.,.).

voluble

Proapoptotic Gene Silencing Via Methylation in Human Tumors,eath, disassembly of various cellular components, and eventual engulfment of the resulting cellular debris (.,.). Inappropriate apoptosis has been associated with a variety of pathological conditions, such as neurodegenerative disorders, autoimmune phenomena, mitochondrial disorders, ischemic damage and cancer (.,.).

obstruct

Book 2005ors in embryonic development, mechanisms of caspase activation, and death receptor mutations in cancer. Additional chapters address death signaling in melanoma, synthetic retinoids and death receptors, the role of p53 in death receptor regulation, immune suppression of cancer, and combination therapy with death ligands.

投票

Book 2005g agonists or antagonists to modulate death signaling for therapeutic purposes. The authors focus on the extrinsic system of death receptors, their regulation and function, and their abnormalities in cancer. Topics of particular interest include resistance to apoptosis, TRAIL signaling, death recept