Titlebook: Damage-Associated Molecular Patternsin Human Diseases; Volume 2: Danger Sig Walter Gottlieb Land Book 2020 Springer Nature Switzerland AG 2

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書目名稱Damage-Associated Molecular Patternsin Human Diseases影響因子(影響力)




書目名稱Damage-Associated Molecular Patternsin Human Diseases影響因子(影響力)學(xué)科排名




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書目名稱Damage-Associated Molecular Patternsin Human Diseases網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Damage-Associated Molecular Patternsin Human Diseases被引頻次




書目名稱Damage-Associated Molecular Patternsin Human Diseases被引頻次學(xué)科排名




書目名稱Damage-Associated Molecular Patternsin Human Diseases年度引用




書目名稱Damage-Associated Molecular Patternsin Human Diseases年度引用學(xué)科排名




書目名稱Damage-Associated Molecular Patternsin Human Diseases讀者反饋




書目名稱Damage-Associated Molecular Patternsin Human Diseases讀者反饋學(xué)科排名

停止償付

seases.Explains how innovative data from immunity and inflam.This book is a continuance of the topic: “DAMPs in Human Diseases”, the basics of which were described in a first volume by the same author. This second volume presents our current understanding of the impact of sterile stress/injury-induc

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Static Portfolio Theory: CAPM and Extensionsof the book .s”: .. Some new aspects of classical and nonclassical cell-associated PRMs and their signaling are presented, by highlighting, for example, the function of supramolecular organizing centers and their role as “prion-like” proteins. The nonclassical PRMs are selectively presented and resu

社團(tuán)

Money, Bonds and Interest Rateslassification of DAMPs as chosen in Vol. 1 is maintained but slightly modified. Thus, four DAMPs categories are defined, termed as (I) endogenous constitutively expressed native DAMPs; (II) endogenous constitutively expressed, injury-modified DAMPs; (III) endogenous inducible DAMPs produced by previ

嘴唇可修剪

New Technology and the Stock Market already comprehensively outlined in Vol. 1 of the book .. At first, the role of stress responses, including autophagic processes, the oxidative stress response, the heat shock response, the unfolded proteins response, and the DNA damage response, is briefly re-examined by focusing on some selected