Titlebook: Cytokines in Severe Sepsis and Septic Shock; Heinz Redl,Günther Schlag Book 1999 Springer Basel AG 1999 Sepsis.clinical trial.cytokine.gen

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Endotoxin scavengers as a therapeutic strategy for sepsishigh mortality rate associated with systemic infections from Gram-negative organisms. Endotoxin remains a viable target for innovative treatment regimens as endotoxin constitutes the most important microbially-derived mediator in septic shock.

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Jila Dana-Haeri,Steven J. Warringtonally absent from the plasma at homeostasis, many cytokines are produced in such large amounts during sepsis that they can be detected in the circulation of the patients. Due to limitation of space, this review will attempt to focus on human sepsis, while mentioning animal models when necessary.

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Possibilities and problems of cytokine measurementsally absent from the plasma at homeostasis, many cytokines are produced in such large amounts during sepsis that they can be detected in the circulation of the patients. Due to limitation of space, this review will attempt to focus on human sepsis, while mentioning animal models when necessary.

CREEK

Nitric oxide and endothelin-1 in circulatory shock involving cytokines and even pathophysiological conditions. Thus, the discovery of two potent vasoactive mediators NO and ET-1 has stimulated an enormous amount of research into the regulation of vascular tone in health and disease.

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Soluble TNF receptors has led many researchers, molecular biologists and clinicians alike, to investigate the complex mechanisms that regulate the production and release of TNF, and that determine its biological effects. The goals of such research efforts are to gain more insight into the pathophysiology of critical ill

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Cytokines, coagulation and fibrinolysiser we will first discuss some aspects of the biochemistry and biology of these systems. Then we will elaborate on the role of cytokines in the activation of clotting and fibrinolysis, in particular during sepsis. Finally, we will summarize possible effects of clotting or fibrinolytic proteins on the

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Apoptosis: Its role in the systemic inflammatory response syndrome and the involvement of cytokines]. Thymocytes that fail to mature with functional receptors are induced to die, thereby limiting the release of none functional cells into the host and preventing autoimmune targeting of the host [3]. During inflammation there is an increased production and accumulation of lymphocytes, monocytes and