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Titlebook: Cytokines and Autoimmune Diseases; Vijay K. Kuchroo,Nora Sarvetnick,Lindsay B. Nichol Book 2002 Springer Science+Business Media New York 2

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發(fā)表于 2025-3-26 23:16:37 | 只看該作者
Yoshiyuki Tomizawa,Ikuo Arai,Shinji Gotohstigators is that MS is a cell-mediated autoimmune disease directed against central nervous system (CNS) myelin and is related in some way to a viral infection (.). Multiple sclerosis means “many scars” and is clearly not an appropriate definition of the disease process. MS is best-defined as “recur
32#
發(fā)表于 2025-3-27 02:57:10 | 只看該作者
33#
發(fā)表于 2025-3-27 06:19:21 | 只看該作者
Edwardo F. Fukushima,Shigeo Hirosely manifestation. This synovial inflammation is commonly followed by a destruction of adjacent cartilage and bone, resulting in the typical erosions that continue one of the diagnotics criteria for RA. Being a common and severe disease, the synovial inflammation in RA has been subject to a continuou
34#
發(fā)表于 2025-3-27 13:26:36 | 只看該作者
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發(fā)表于 2025-3-27 15:14:18 | 只看該作者
36#
發(fā)表于 2025-3-27 20:50:07 | 只看該作者
Federico Bussolino,Alberto Mantovanih) cells to generate autoantigen-reactive pathogenic or protective effectors, they help self-reactive B cells to produce autoantibodies, and they also participate in mediating tissue damage in the target organ. Cytokines also control tissue tolerance and “immune-privilege” and prevent the propagatio
37#
發(fā)表于 2025-3-27 23:34:43 | 只看該作者
Book 2002he pathogenesis of autoimmune diseases. The many chapters offer critical reviews the basic mechanisms controlling cytokine induction and regulation, as well as the resulting production of proinflammatory and anti-inflammatory cytokines, the former of which induces organ-specific autoimmune diseases.
38#
發(fā)表于 2025-3-28 05:43:54 | 只看該作者
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發(fā)表于 2025-3-28 08:26:28 | 只看該作者
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發(fā)表于 2025-3-28 13:14:57 | 只看該作者
Role of Cytokines in Multiple Sclerosis and in Mucosal Tolerancerent inflammation of CNS white matter leading to myelin destruction and progressive neurologic impairment.” The major question that then must be addressed is what causes this specific and recurrent inflammation and how can it be regulated.
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