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Titlebook: Current Topics in Innate Immunity II; John D. Lambris,George Hajishengallis Book 2012 The Editor(s) (if applicable) and The Author(s), und

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書(shū)目名稱(chēng)Current Topics in Innate Immunity II
編輯John D. Lambris,George Hajishengallis
視頻videohttp://file.papertrans.cn/242/241359/241359.mp4
概述Summary of latest Aegean Conferences meeting - the 7th Innate Immunity Conference.Brings together findings of international scientists with different advanced perspectives.Discusses Evolution of the I
叢書(shū)名稱(chēng)Advances in Experimental Medicine and Biology
圖書(shū)封面Titlebook: Current Topics in Innate Immunity II;  John D. Lambris,George Hajishengallis Book 2012 The Editor(s) (if applicable) and The Author(s), und
描述Mounting evidence in the past decade indicates that innate immunity mediates functions above and beyond first-line defense against infection.It is now appreciated that innate immune mechanisms are critically involved in the development of adaptive immunity and, moreover, the regulation of diverse physiological and homeostatic processes. The latter explains why deregulation of innate immunity may lead to pathological disorders that are not necessarily or directly related to host defense. This Volume compiles the latest advances in this rapidly evolving field as presented by eminent scientists at the 7th International Aegean Conference on Innate Immunity in Rhodes, Greece.It includes topics related to the biology and function of Toll-like and other pattern-recognition receptors, complement and its crosstalk with other physiological systems, inflammatory mechanisms and diseases, natural killer cells, and the cooperative interplay between innate and adaptive immune cells.This book is an excellent source of information for researchers and clinicians with interests in immunology, host-microbe interactions, and infectious and inflammatory diseases.
出版日期Book 2012
關(guān)鍵詞infectious diseases
版次1
doihttps://doi.org/10.1007/978-1-4614-0106-3
isbn_softcover978-1-4614-2952-4
isbn_ebook978-1-4614-0106-3Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
The information of publication is updating

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,The Role of Innate Immunity in Trafficking of Hematopoietic Stem Cells—An Emerging Link Between Actwith sphingosine-1 phosphate (S1P) orchestrate HSPC mobilization. On other hand some other bioactive lipids e.g., ceramide-1-phosphate (C1P) that is released from damaged/“l(fā)eaky” cells in BM after myeloablative conditioning for transplant may play an opposite important role in homing of HSPCs to BM.
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Role of C3, C5 and Anaphylatoxin Receptors in Acute Lung Injury and in Sepsis,ress inflammatory responses or divert them from Th1 to Th2 responses, impacting the innate immune system. Finally, in experimental polymicrobial sepsis, there is evidence that many of the adverse outcomes can be linked to the roles of C5a and engagement of its two receptors, C5aR and C5L2. These obs
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Complement Involvement in Neovascular Ocular Diseases,ogenic inhibitors. However, these treatments do not address the underlying cause of many of these diseases. A clear understanding of the cellular and molecular mechanisms could bring a major shift in our approach to disease treatment and prevention.
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發(fā)表于 2025-3-22 14:30:36 | 只看該作者
Platelets, Complement, and Contact Activation: Partners in Inflammation and Thrombosis,onsible for recruiting soluble regulators to the surface. Platelet-bound C3(H.O) acts as a ligand for leukocyte CR1 (CD35), potentially enabling platelet–leukocyte interactions. In addition, platelet activation leads to the activation of contact system enzymes, which are specifically inhibited by an
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Herrn Hannes Stoppel,Frau Birgit Griesewith sphingosine-1 phosphate (S1P) orchestrate HSPC mobilization. On other hand some other bioactive lipids e.g., ceramide-1-phosphate (C1P) that is released from damaged/“l(fā)eaky” cells in BM after myeloablative conditioning for transplant may play an opposite important role in homing of HSPCs to BM.
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https://doi.org/10.1007/978-3-322-97057-2han one hour after trauma, Injury Severity Score?>?18, .?=?5) presented a significant increase in granzmye B levels in neutrophils and lymphocytes. Thus, tissue trauma is associated with early activation of both, the complement and apoptosis system. The present data suggest a new form of interaction
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