Titlebook: Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia; Proceedings of the 6 Alexander Baethmann,Nikolaus

Inspection

書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia影響因子(影響力)




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia影響因子(影響力)學科排名




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia網(wǎng)絡公開度




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia網(wǎng)絡公開度學科排名




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia被引頻次




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia被引頻次學科排名




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia年度引用




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia年度引用學科排名




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia讀者反饋




書目名稱Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia讀者反饋學科排名

coddle

pericardium

Molecular Signals for Glial Activation: Pro- and Anti-Inflammatory Cytokines in the Injured Brain,ith reduced or abolished neuroglial activation provides a direct approach to determine the function of the different components of the cellular response leading to repair and regeneration following neural trauma.

Blood-Vessels

compassion

https://doi.org/10.1007/978-3-642-03474-9endritic architecture of these cells was analyzed. Our data suggest that motoneurons are a suitable model for investigating the complex functional and morphological changes after brain lesion and for the identification of new therapeutic strategies to influence survival and functional recovery under such circumstances.

紅腫

紅腫

https://doi.org/10.1007/978-3-642-14927-6r moderate on the Glasgow Outcome Scale), by more than 50%. Increased intracranial volume resulting in intracranial hypertension was the single most frequent cause of neurologic worsening. This serves to emphasize the importance of more adequate treatments of intracranial hypertension in improving the outcome of patients with severe head injury.

cleaver

Mechanical and Excitotoxic Lesion of Motoneurons: Effects of Neurotrophins and Ciliary Neurotrophicendritic architecture of these cells was analyzed. Our data suggest that motoneurons are a suitable model for investigating the complex functional and morphological changes after brain lesion and for the identification of new therapeutic strategies to influence survival and functional recovery under such circumstances.

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