Titlebook: Current Concepts in Autoimmunity and Chronic Inflammation; Andreas Radbruch,Peter E. Lipsky Book 2006 The Editor(s) (if applicable) and Th

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Introduction: Writing the Holy Land, a functionally mature T cell subpopulation specialized for suppressive function. Their generation is in part genetically and developmentally controlled. Genetically determined or environmentally induced abnormality in CD25.CD4. regulatory T cell development, maintenance, and function can be a cause of autoimmune disease in humans.

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https://doi.org/10.1007/978-3-030-52988-8vant tissue damage. Recent experimental evidence has demonstrated that both the induction of autoreactivity and the conversion into autoimmune disease is controlled by the activation of the nonspecific innate immune system. Also, the “inflammatory status” of the target organ critically influences the onset of overt autoimmune disease.

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Chien-Liang Chen,Ding-Jung Chiangh it may provoke a prolonged inflammatory response when occurring on a susceptible immunological background. The argument of infective causality is by nomeans trivial, since it carries important consequences for the safety of vaccine development.

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Altug Yalcintas,Eylül Seren K?seln lead to the development and propagation of autoimmune diseases such as lupus erythematosus or chronic inflammatory rheumatoid arthritis by the autoantibody BcR-expressing B cells and their secreted autoantibodies.

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