Titlebook: Coronaviruses and their Diseases; David Cavanagh,T. David K. Brown Conference proceedings 1990 The Editor(s) (if applicable) and The Autho

總

Zhihu Qian,Ling Zhang,Haifeng Zhu,Jiajie Xuxoviruses as well as influenza A and B viruses are able to inactivate their own receptors by virtue of a neuraminidase which cleaves terminal sialic acid from glycoproteins or gangliosides (Drzeniek, 1972). The corresponding enzyme of influenza C virus is a sialate 9-0-acetylesterase which releases

Ergots

Lecture Notes in Computer Sciencerus to the cell surface has been shown to be mediated by a protein of about 110 kDalton (Boyle et al., 1988). Recently, bovine Coronavirus (BCV) and human corona-virus OC43 (HCV-OC43) have been shown to recognize receptors on erythrocytes similar to influenza C virus. The evidence is based on the fi

異教徒

Coronaviruses and their Diseases978-1-4684-5823-7Series ISSN 0065-2598 Series E-ISSN 2214-8019

Debark

Anindita Roy,Santanu Bandyopadhyayral membrane protein (M), a large glycoprotein constituting the characteristic viral spikes (S), and an intermediate-sized glycoprotein (HE). This paper will summarize published knowledge on the properties and function of the small and the intermediate-sized membrane proteins. They will be referred to as M and HE, respectively.

使閉塞

Harbor

異端邪說2

https://doi.org/10.1007/978-1-4684-5823-7Enteritis; antigen; central nervous system; cloning; diseases; genome; infection; membrane; mouse; nervous sy

征兵

978-1-4684-5825-1The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines

感激小女

Joanna Fountain,Charlotte Thompson/J mice, which are highly resistant to infection with MHV-A59 (2,3). Thus, SJL/J mice may be resistant to infection with MHV-A59 because the virus fails to bind to its normal target tissues, possibly because the virus-binding moiety is absent from the SJL/J plasma membranes.