Titlebook: Coronary Tone in Ischemic Heart Disease; Wolfram Kupper,Rex N. MacAlpin,Walter Bleifeld Book 1984 Springer Science+Business Media Dordrech

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Enhanced transcardiac 1-norepinephrine response during cold pressor test in obstructive coronary ar. It became apparent that alpha adrenergic mediated coronary vasoconstriction can compete with metabolically-induced coronary vasodilatation, particularly in myocardial regions with decreased coronary reserve [1–7]. Recent studies have also emphasized that increased sympathetic nervous activity enha

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Dietary Prevention of Coronary Heart Diseaseobstruction or a spastic vasoconstriction of an epicardial coronary artery. Both of these mechanism do not sufficiently explain the variable clinical syndromes of angina pectoris, particularly not the variation in anginal threshold experienced by many patients. Therefore, the concept of a variable v

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Deborah M. Bethards MD,Ann Ouyang MBBSdetermine by direct and continuous measurements, the extent to which activation of β. and β. adrenergic receptors elicited vasodilation of large and small coronary vessels in conscious dogs. This was accomplished by administering isoproterenol, an agent which contains β. and β. adrenergic properties

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Puberty Timing Remains Unchanged stimulation. Some clinical reports [4–7] have strongly suggested that in patients with variant angina alpha-adrenergic stimulation could induce clinical episodes of coronary spasm, whereas alpha-adrenergic blockade could be effective in alleviating these episodes. Thus, the alpha-adrenergic nervous

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Peter A. Lee MD, PhD,Christopher P. Houk MD patients with coronary artery disease. The alpha-adrenergic nervous system may be potential physiologic mediator of such inappropriate coronary artery vasoconstriction. Animal studies have documented that tonic alpha-adrenergic mediated vasoconstriction tone exists both at rest and with exercise [1

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Cervical Cancer Detection and Prevention,e effectiveness of these compounds has been related to the reduction in oxygen demand due to the attenuated myocardial response to sympathetic stimuli [2]. However, in patients with variant angina there has been some concern that beta-blockade promotes coronary artery spasm [10]. Recent reports reve