Titlebook: Coronary Microvascular Dysfunction; Filippo Crea,Gaetano A. Lanza,Paolo G. Camici Book 2014 The Editor(s) (if applicable) and The Author(s

Enkephalin

書目名稱Coronary Microvascular Dysfunction影響因子(影響力)




書目名稱Coronary Microvascular Dysfunction影響因子(影響力)學(xué)科排名




書目名稱Coronary Microvascular Dysfunction網(wǎng)絡(luò)公開度




書目名稱Coronary Microvascular Dysfunction網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Coronary Microvascular Dysfunction被引頻次




書目名稱Coronary Microvascular Dysfunction被引頻次學(xué)科排名




書目名稱Coronary Microvascular Dysfunction年度引用




書目名稱Coronary Microvascular Dysfunction年度引用學(xué)科排名




書目名稱Coronary Microvascular Dysfunction讀者反饋




書目名稱Coronary Microvascular Dysfunction讀者反饋學(xué)科排名

constitute

騎師

CMD in Myocardial Diseases mainly driven by sudden death, probably caused by myocardial necrosis-related ventricular arrhythmias. This prognostic information is additive to that provided by traditional risk factors and can therefore improve the identification of patients who need ICD implantation.

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人充滿活力

Treatment of CMD in Myocardial Diseasesremains to be established. There are no studies assessing the effects of therapies in aortic stenosis, myocarditis, and amyloidosis, whereas treatment with alpha galactosidase A failed to improve CMD in Anderson-Fabry’s disease, despite achieving a significant improvement of symptoms and clinical conditions.

floaters

floaters

Respiratory Metabolism in Wheat angina syndrome, but can also occur as an acute syndrome suggesting a non-ST elevation ACS. Takotsubo cardiomyopathy is likely also caused by a sudden acute vasoconstriction of resistive coronary arteries resulting in severe impairment of med-distal LV segments.

archenemy

Systemic

Introduction to occupational health,this chapter. While the pharmacological approach to acute MVA still waits to be defined, treatment of takotsubo disease is mainly based on the control of hemodynamic state, due to the usual spontaneous improvement of the disease.

jet-lag

Health promotion in the workplace,cluding pharmacological (e.g., adenosine, nitroprusside) and nonpharmacological (e.g., antiembolic devices, ischemic conditioning) tools. This chapter discusses the main results observed with these interventions in these clinical settings.