Titlebook: Coronary Circulation in Physiological and Pathophysiological States; Motoomi Nakamura,Paul M. Vanhoutte Book 1991 Springer-Verlag Tokyo 19

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Effects of Atherosclerosis and Regression of Lesions,tentiation of vasoconstrictor responses to thromboxane and endothelin, and modest impairment of vasodilator responses to ADP. Our studies in vitro and several others indicate that endothelium-dependent relaxation is impaired by atherosclerosis. It is likely but not proven, that impairment of endothe

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Book 1991orly understood. It has long been thought that ischemic heart disease is the sequence of an organic fixed atherosclerotic obstruction of the epicardial coronary arteries and the role of coronary vasomotion has been given much less attention. Recent clinical and laboratory animal studies revealed tha

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brachial-plexus

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Effects of Native and Oxidized Low Density Lipoproteins on Formation and Inactivation of EDRF and Vn, EDRF-activity, and vascular smooth muscle tone. Native (n) LDL was isolated from fresh human plasma via sequential ultracentrifugation, and oxidized by Cu.-incubation. EDRF released from cultured endothelial cells was inactivated by both n-LDL and ox-LDL (1 mg/ml), as detected in a bioassay syste

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Endothelium-Derived Vasoactive Factors, Platelets and Coronary Disease,ich most likely is nitric oxide, or a nitrosoderivative releasing nitric oxide, derived from 1-arginine), and endothelium-derived hyperpolarizing factor (EDHF, which possibly is a labile metabolite of arachidonic acid formed through the P-450 pathway). Possible endothelium-derived contracting factor