Titlebook: Contraception; Donna Shoupe (Associate Professor),Florence P. Has Book 1993 Springer-Verlag New York, Inc. 1993 AIDS.Fortpflanzung.Geburte

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https://doi.org/10.1007/978-3-531-91193-9in humankind’s never ending quest for securing an adequate supply of food. In this chapter we principally discuss contraceptive steroids; adrenal, androgenic, and anabolic hormones are mentioned only to supplement the general picture. Milestones of steroid development are given in Table 1–1.

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Pharmacology of Contraceptive Steroids,s (norethindrone and norethynodrel) were contaminated during the manufacturing process with an estrogen, mestranol — the 3-methyl ether of ethynyl estradiol. The degree of contamination, 0.15%, appeared trivial to the chemists, but meant that every clinical dose of 10 mg norethindrone (that was the

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Oral Contraceptives Effect on Glucose Metabolism, United States in 1960. They were actively marketed and sales increased dramatically in the early years of that decade to move oral contraceptives into a leading position in methods used for family planning. The initial drugs contained a high dosage of synthetic estrogens and progestins; for example

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Oral Contraceptives and Plasma Lipoprotein Metabolism,ipoprotein metabolism are potentially important.. Commonly used OC preparations result in elevations in plasma triglycerides (TG), very low-density lipoproteins (VLDL) and low-density lipoproteins (LDL), and reductions in high-density lipoproteins (HDL), particularly the HDL. subclass. In this chapt

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Oral Contraceptives: Effect on Hemostasis,ge between sex steroids and abnormalities in hemostasis.. A series of epidemiologic studies was subsequently published — primarily from the United Kingdom — which supported this initial clinical observation, and it was concluded that women using oral contraceptives were at increased risk for venous