Titlebook: Concise Guide to Hematology; Hillard M. Lazarus,Alvin H. Schmaier Book 2019Latest edition Springer Nature Switzerland AG 2019 hematopoiesi

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https://doi.org/10.1007/978-981-13-6864-6 an explanation of the pathophysiology of iron depletion. The phases of the development of iron-deficiency anemia and its prevalence in different populations at risk are outlined. The clinical manifestations of iron deficiency and the resulting anemia are reviewed, along with a description of labora

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Traces of Violence and Freedom of Thoughtia are the most common inherited hemoglobinopathies. The clinical phenotype of these hemolytic disorders are contingent on the number and type of mutations or deletions inherited. Both qualitative and quantitative disorders occur. Understanding the genetic basis for these disorders facilitates repro

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The Influence of Glacial Meltwater,ally by increased reticulocytosis. In contrast to hereditary hemolytic anemias, the decreased erythrocyte survival due to increased red blood cell destruction is caused typically by extrinsic factors including immune-mediated mechanisms (autoimmune or alloimmune causes), mechanical injury to erythro

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K. Gdanietz,K. Vorpahl,G. Piehl,A. H?ckof fibrinogen; prothrombin; factors V, VII, X, XI, and XIII; and PAI-1 constitute rare bleeding disorders (RBDs). Inherited deficiencies of inhibitors of fibrinolysis are extremely rare bleeding disorders and are included under the “miscellaneous” category. The clinical spectrum of bleeding symptoms

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https://doi.org/10.1007/978-3-642-71665-2e coagulation tests, such as the platelet count, prothrombin time, or activated partial thromboplastin time. Acquired bleeding disorders can be categorized in quantitative or qualitative platelet defects, impaired clotting factor synthesis or low coagulation factors caused by inhibiting antibodies,

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https://doi.org/10.1007/978-3-662-42322-6ng events involving the blood vessel, platelets, and plasma coagulation system. Platelets are anucleate fragments derived from megakaryocytes and have major roles in hemostasis. This chapter focuses on platelet structure and function and on inherited disorders of platelet number and function, which

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https://doi.org/10.1007/978-3-319-67867-2ative), sequestration (hypersplenism), increased platelet consumption, and increased platelet destruction. Consumptive thrombocytopenic disorders include thrombin-mediated (disseminated intravascular coagulation), septicemia, and thrombotic microangiopathy (thrombotic thrombocytopenic purpura, hemol