Titlebook: Concepts in Viral Pathogenesis; Abner Louis Notkins,Michael B. A. Oldstone Textbook 1984 Springer-Verlag New York Inc. 1984 Antigen.Hepati

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Double Semion Model as a Quantum Memorywn that both virus and host-cell receptors are genetically controlled [1,2]. An excellent review of general principles related to animal virus attachment has been written recently by Holmes [3]. A key issue in relating host-cell molecules to virus attachment is identification of the “productive rece

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Topological Orders with Spins and Fermionsed by later replicative stages. ADE is thus conditional upon the presence on host cells of the necessary accessory receptors, but it is also determined by the nature of the infecting virus, by the specificity of the antiviral antibodies, and by the conditions under which the cells, virus, and antibo

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Viral Genes and Tissue Tropismerent tissues, as well as to different cells within those tissues, results in distinct patterns of infectious diseases [2]. Recent studies with the mammalian reoviruses and their infection of newborn mice have identified the specific viral protein that determines tropism (the viral hemagglutinin) an

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Utilization of Host Proteins as Virus Receptorsologic research has been devoted to describing strategies used by different viruses to survive despite attempts by the host to prevent their replication. One hypothesis holds that virus attachment to host cell molecules unique to susceptible cells governs virus spread to, and infection of, specific

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Host Range and Tissue Tropisms: Antibody-Dependent Mechanismstes of appropriate viral specificity, followed by stages of penetration, uncoating, and replication of the viral genome. With most infections, antiviral antibody will inhibit the initial binding step, and in so doing, will reduce viral infectivity. However, in certain cell—virus interactions, the pr

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