Titlebook: Computational Cardiovascular Mechanics; Modeling and Applica Julius M. Guccione,Ghassan S. Kassab,Mark B. Ratcl Book 2010 Springer-Verlag U

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epicardium

終止

https://doi.org/10.1007/BFb0118897sis in coronary heart disease since lesions are preferentially located at bifurcation points and regions of high curvature. The influence of these local structures on recirculation and deleterious shear stresses and their role in plaque development is widely accepted. Over time, researchers have tur

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https://doi.org/10.1007/BFb0118844I techniques: the arbitrary Lagrangian Eulerian (ALE) and the immersed boundary (IB) methods. Examples of FSI applications to aortic cross-clamping used during surgical treatment of heart failure and valveless pumping are also presented.

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https://doi.org/10.1007/BFb0118844s known to have adverse effects on the function of the cardiovascular (CV) system. This chapter outlines the basic features of turbulence in the CV system. As a specific example, simulation of turbulent flow in an abdominal aortic aneurysm (AAA) is presented. The simulated results show that transiti

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Spectra of 3d five-coordinate complexes,pe, size, and function of the LV are caused by altered mechanical properties of the injured myocardium. As the survival rate after MI improves with medical advances, the incidence of HF patients increases. Hence, an accurate depiction of the LV remodeling process facilitates disease surveillance and

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https://doi.org/10.1007/BFb0118851vel surgical procedure or medical device for treating heart failure or ischemic cardiomyopathy. In each study our cardiac biomechanics laboratory uses one of two different approaches: (1) an axisymmetric truncated ellipsoidal model with left ventricular (LV) cavity and wall volumes typical of the fa

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Reversible Oxygenierung von Metallkomplexen,echanism is presumed to be positive feedback between diastolic LV wall stress and eccentric LV hypertrophy. Further, in each case, an increase in LV size is an important adverse prognostic finding. The experience with skeletal muscle cardiomyoplasty led to the hypothesis that passive constraint of L