Titlebook: Cocaine and Other Stimulants; Everett H. Ellinwood,M. Marlyne Kilbey Book 1977 Springer Science+Business Media New York 1977 Methylphenida

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Changes in Neuronal Activity in the Neostriatum and Reticular Formation Following Acute or Long-Ter, this area may contribute to the expression of amphetamine-induced electroencephalographic arousal. This view is consistent with the wide variety of evidence suggesting that the reticular formation of the brainstem mediates electroencephalographic and behavioral arousal (e.g., Moruzzi and Magoun, 1

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Amygdala Hyperspindling and Seizures Induced by Cocaine,(at times the animal appears to be reacting to non-existent stimuli) are often immediately preceded by a high voltage spindle and/or discharge in the amygdala, accumbens, and olfactory tubercle (Ellinwood, 1974a; Ellinwood, Sudilovsky, and Nelson, 1974). Much lower doses of amphetamine (10 mg/kg) wh

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Sensitization to Cocaine Following Chronic Administration in the Rat,ndell, 1974). This sensitization and its underlying mechanisms are of particular interest because of their possible relationship to the gradual evolution of psychosis seen in humans during chronic use of these drugs (Ellinwood, 1967; Connell, 1958; Lewin, 1931; Ellinwood, Sudilovsky, and Nelson, 197

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https://doi.org/10.1007/978-94-009-5024-5s are in definitions and methodology. It is, therefore, our intention in this chapter to examine the literature related to the site of stimulant drug action in the brain, with particular reference to amphetamine and apomorphine and the stereotyped behaviour patterns and hyperactivity induced by thes

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https://doi.org/10.1007/978-3-642-56267-9 in the nerve terminals. On the other hand, the central stimulant actions of methylphenidate and cocaine are reduced by reserpine pretreatment but not by α-methyltyrosine (van Rossum, van der Schoot, and Hurkmans, 1962; Scheel-Krüger, 1971; Simon, Sultan, Chermat, and Boissier, 1972) suggesting that

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Shelly Sheats Harkness,Catherine Lane of many processes are involved, including increased neurotransmitter synthesis, altered receptor sensitivity, and activation of inhibitory serotonergic pathways (Koda and Gibb, 1973; Mandell and Morgan, 1970; Sparber and Tilson, 1972).

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Shaun Stearns,Charles C. Benight, this area may contribute to the expression of amphetamine-induced electroencephalographic arousal. This view is consistent with the wide variety of evidence suggesting that the reticular formation of the brainstem mediates electroencephalographic and behavioral arousal (e.g., Moruzzi and Magoun, 1