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Titlebook: Clinical efficacy of positron emission tomography; Proceedings of a wor W.-D. Heiss,G. Pawlik,K. Wienhard Conference proceedings 1987 Sprin

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書目名稱Clinical efficacy of positron emission tomography
副標(biāo)題Proceedings of a wor
編輯W.-D. Heiss,G. Pawlik,K. Wienhard
視頻videohttp://file.papertrans.cn/229/228308/228308.mp4
叢書名稱Developments in Nuclear Medicine
圖書封面Titlebook: Clinical efficacy of positron emission tomography; Proceedings of a wor W.-D. Heiss,G. Pawlik,K. Wienhard Conference proceedings 1987 Sprin
描述The series of workshops sponsored by the European Communities started with "Methodology of PET" at Hammersmith Hospital, London, in March 1984. This was followed by "Radiochemistry, Methodology and Standardization in PET" at the Service Hospitalier Frederic Joliot in Orsay, France, in March 1985. Both these meetings were, in the opinion of all participants, great successes, and it was agreed that such work- shops should continue and be organized on the same basis. After these two workshops on the fundamentals of PET, time now is ripe to evaluate the clinical efficacy of PET investigations, and to discuss to what extend the information provided by this high technology and theoretical area has contributed to the understanding of disease mechanisms, leading to immediate clinical applications. As pointed out in the previous meetings, PET using short-lived radioisotopes produced in an on-line cyclotron is restricted to a few centers. Therefore, the topics studied so far were mainly of scientific interest and clinical problems were dealt with only marginally. Before this costly technique can be spread and new information made accessible to a broader clinical clientele, its clinical value
出版日期Conference proceedings 1987
關(guān)鍵詞Public Health; Tumor; brain; diseases; heart; magnetic resonance imaging (MRI); neoplasm; neurology; oncolog
版次1
doihttps://doi.org/10.1007/978-94-009-3345-3
isbn_softcover978-94-010-8002-6
isbn_ebook978-94-009-3345-3
copyrightSpringer Science+Business Media Dordrecht 1987
The information of publication is updating

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Ischemic Stroke Studied by 15O-Labeled Compounds: Misery Perfusion and Luxury Perfusion understanding of the pathophysiological mechanisms of tissue damage and dysfunction in acute stroke patients (Ackerman et al., 1981; Baron, 1985; Frackowiak, 1985; Phelps et al., 1982; Powers and Raichle, 1985).
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地板
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Pet Studies in Internal Carotid Occlusion Patients: Base-Line Hemodynamic and Metabolic Alterations;omographic studies of patients with internal carotid artery (ICA) occlusion (Baron et al., 1981; Gibbs et al., 1984, 1985; Samson et al., 1985a,b; Powers et al., 1984a, 1984b). This paper summarizes our findings in such patients. For practical purposes, focal and bilateral (diffuse) functional abnor
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發(fā)表于 2025-3-22 14:53:34 | 只看該作者
Positron Emission Tomography in Cerebrovascular Disorders: Results and Perspectiveshen it correctly channels a treatment. Neuroimaging techniques are, in this respect, a classical example. The best known technique, the CT scan, is in fact relevant for the identification, for the management and for the follow-up of many organic brain syndromes, from tumors to hydrocephalus to cereb
7#
發(fā)表于 2025-3-22 19:29:40 | 只看該作者
Changes of Cerebral Glucose Metabolism in Movement Disorders technique. For a comprehensive review of PET findings in movement disorders, including changes in flow, oxygen and glucose metabolism, as well as neurotransmitter changes, the reader is referred to a recently published monograph (Leenders, 1986).
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發(fā)表于 2025-3-23 05:21:16 | 只看該作者
SUMMARY of the Discussion on Brain Ihysiological tracers to be used in various pathological conditions such as cerebro-vascular diseases. All the data presented also have stressed the non-invasive aspect of PET investigations which allow to open many windows in the early diagnosis and the follow-up of treatment of patients with brain
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發(fā)表于 2025-3-23 08:57:43 | 只看該作者
Localizing Epileptic Foci by PETstructures which were involved in the pathological activity. Even in focal seizures this activity and the increase of metabolism was frequently spread intracortically, transcallosally, and to basal structures (Collins et al., 1976). The increase of metabolism appears to be mainly due to the metaboli
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