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Titlebook: Clinical Pathology of Pancreatic Disorders; John A. Lott Book 1997 Humana Press Inc. 1997 Laboratory.biochemistry.chemistry.diabetes.diagn

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發(fā)表于 2025-3-21 19:50:38 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Clinical Pathology of Pancreatic Disorders
編輯John A. Lott
視頻videohttp://file.papertrans.cn/229/228149/228149.mp4
叢書名稱Pathology and Laboratory Medicine
圖書封面Titlebook: Clinical Pathology of Pancreatic Disorders;  John A. Lott Book 1997 Humana Press Inc. 1997 Laboratory.biochemistry.chemistry.diabetes.diagn
描述The pancreas is about the size and shape of the hand; the tail points to the spleen, and the head is nestled in a loop of the duodenum. Loss of the exocrine (digestive) func- tions commonly leads to severe gastrointestinal disturbances, malabsorption, a cata- bolic state, and weight loss in the face of an adequate diet. Loss of endocrine pancreatic function leads to a large spectrum of disorders associated with the loss of hormone secretions; the most common and most severe is diabetes mellitus. Loss of the entire pancreas owing to trauma, surgery, atherosclerosis, or other medical problems leaves the patient in a digestive and metabolic crisis. The correct diagnosis of pancreatic disorders remains a challenge given the multi- faceted function of the pancreas. The clinical laboratory plays an important role, and other tools such as CAT scans, ultrasound, radiographs, biopsies, and even surgery are used to make a diagnosis. The emphasis of Clinical Pathology of Pancreatic Disorders is on the clinical laboratory definition of pancreatic pathology. Disorders of the endocrine pancreas can be highly complex, and sophisticated tests are needed to determine the nature of the disease, its
出版日期Book 1997
關(guān)鍵詞Laboratory; biochemistry; chemistry; diabetes; diagnosis; diseases; inflammation; management; monitoring; pan
版次1
doihttps://doi.org/10.1007/978-1-4612-3964-2
isbn_softcover978-1-4612-8443-7
isbn_ebook978-1-4612-3964-2
copyrightHumana Press Inc. 1997
The information of publication is updating

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978-1-4612-8443-7Humana Press Inc. 1997
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ST-Streckenanalyse im Langzeit-EKGude cystic fibrosis, atrophic pancreatitis, chronic pancreatitis, infarction of the pancreas, surgical removal of the gland, atherosclerosis of the pancreas, pancreatic cysts, and other miscellaneous causes. More than about 85% of the acinar cells must be lost before abnormalities appear, indicating
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On Multipartition Communication Complexityamylase activity in blood and urine was a sensitive and reliable test for various pancreatic disorders. Since this time, numerous other enzymatic markers of pancreatic disease have been described. Although there is a large body of literature indicating that some of these other tests may provide bett
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A Space Lower Bound for Routing in Treestalities, although prognosis is dependent on histopathological type. Early and accurate diagnosis improves survival, but is difficult. Pancreatic neoplasms may be classified as tumors of the exocrine pancreas, tumors of the endocrine pancreas, and atypical neoplasms, such as lymphomas, mesotheliomas
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The Boolean Hierarchy of NP-Partitions,rated by connective tissue. These cells constitute almost 80% of the organ. An acinus consists of up to 50 cells that have a common orientation toward a central lumen. Each acinus is drained by a ductule.
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