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Titlebook: Clinical Handbook of Cardiac Electrophysiology; Benedict M. Glover,Pedro Brugada Book 2021Latest edition Springer Nature Switzerland AG 20

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發(fā)表于 2025-3-25 06:58:37 | 只看該作者
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發(fā)表于 2025-3-25 11:15:56 | 只看該作者
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發(fā)表于 2025-3-25 14:20:32 | 只看該作者
Cardiac Anatomy and Electrophysiology, mapping and substrate modification. Training in cardiac electrophysiology requires a detailed knowledge of the anatomy and physiology of the heart. In order to understand the basis of cardiac electrophysiology it is important to discuss the different phases of the cardiac action potential, variabil
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發(fā)表于 2025-3-25 17:38:51 | 只看該作者
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發(fā)表于 2025-3-25 23:20:39 | 只看該作者
Electroanatomic Mapping,ncompassing this data. These systems use magnetic, impedance or a combination of the two for the non-fluoroscopic location of the catheters. Data can be displayed on a recreated anatomic structure. This data includes activation times, voltage recordings and entrainment mapping. One of the original m
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發(fā)表于 2025-3-26 03:00:44 | 只看該作者
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發(fā)表于 2025-3-26 06:44:33 | 只看該作者
Accessory Pathway (AP) Conduction,h the baseline ECG may be normal, the common features seen are a slurring of the initial portion of the QRS complex resulting in a delta wave, a short PR interval during normal sinus rhythm, a wide QRS duration, and secondary ST and T wave changes, all of which result from a combination of eccentric
28#
發(fā)表于 2025-3-26 10:45:43 | 只看該作者
Atrial Tachycardias,ximately 7% of all SVT’s (Wellens HJ, Circulation. 90(3): 1576–1577, 1994)..Focal atrial tachycardia occurs as a result of either micro re-entry, increased automaticity or triggered activity while macro re-entry occurs over a region of tissue surrounding a region of conduction block. Focal atrial ta
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發(fā)表于 2025-3-26 16:13:27 | 只看該作者
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發(fā)表于 2025-3-26 19:40:10 | 只看該作者
Atrial Fibrillation,ica (Chugh et al., Circulation 129:837–847, 2014). In addition to causing considerable adverse sequelae and an increase in hospitalizations, there is a five-fold increase in the risk of stroke associated with non-valvular AF (Wolf et al., Stroke 22:983–988, 1991) which increases by a factor of 17 in
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