Titlebook: Clinical Aspects of O2 Transport and Tissue Oxygenation; K. Reinhart,K. Eyrich Conference proceedings 1989 Springer-Verlag Berlin Heidelbe

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Mechanisms and Modifications of Hyperoxic Injury to the Mammalian Pulmonary Surfactant System The increased permeability of the blood-gas barrier to solute and solvent leads to the formation of noncardiogenic interstitial and alveolar edema, atelectasis, hypoxemia, and hypercapnia, and causes the death of the animals from hypoxemia or respiratory acidosis [2, 6].

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Interaction Between Circulating Granulocytes and Xanthine Oxidase-Derived Oxidants in the Postischemverted to the highly cytotoxic hydroxyl radical by the iron catalyzed Haber-Weiss reaction. This initiates the process of lipid peroxidation which may stimulate the release of chemoattractants and the subsequent activation and recruitment of granulocytes.

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Clinical Aspects of O2 Transport and Tissue Oxygenation978-3-642-83872-9

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Mechanisms Regulating the Reactions of Human Hemoglobin with Oxygen and Carbon Monoxidel mechanism of hemoglobin has come from the study of abnormal human hemoglobins. Now that the genes for the α and . chains of hemoglobin and for the single chain of myoglobin have been cloned in . directed mutagenesis has provided new tools for probing the reactions of these proteins with ligands. F

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Circulatory Compensation as a Response to Hypoxiaes, and there are local (often competing) vascular responses. The circulatory compensation to hypoxia, then, is not a simple, single response but the integration of all these variables. The use of laboratory animal models offers the chance to open the control loops involved and to analyze their comp

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Supply Dependency of Oxygen Uptaken the essential differences between normal or physiological O. supply dependency and its abnormal or pathological manifestation. Following that, I will present some evidence that the pathological form is a very bad prognostic sign. Finally, I will report the results from some animal experiments in w

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Physiological and Biochemical Indicators of Impaired Tissue Oxygenation the environment. The more complex the life form, the more likely the possibility that under certain circumstances sufficient O. would not be available everywhere in the body to maintain adequate energy supplies using aerobic metabolic pathways. This required the persistance of anaerobic means of en

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https://doi.org/10.1007/978-3-319-68792-6l mechanism of hemoglobin has come from the study of abnormal human hemoglobins. Now that the genes for the α and . chains of hemoglobin and for the single chain of myoglobin have been cloned in . directed mutagenesis has provided new tools for probing the reactions of these proteins with ligands. F