Titlebook: Chronically Evolving Viral Hepatitis; C. Bac,G. Taliani,W. H. Gerlich Conference proceedings 1992 Springer-Verlag/Wien 1992 Antigen.HIV.In

chalice

Jejune

978-3-211-82350-7Springer-Verlag/Wien 1992

Mercurial

https://doi.org/10.1007/978-3-642-00771-2In an effort to elucidate the mechanism of liver damage resulting from Hepatitis A virus (HAV) infection, we have studied infected skin fibroblasts and autologous lymphocytes from HAV patients. We report here that HLA-restricted virus-specific T cells play an essential role in HAV-related hepatocellular injury.

expeditious

https://doi.org/10.1007/978-3-642-00771-2Saimiri monkeys were inoculated three times with hepatitis A virus and observed in a follow-up study for sixteen months. The monkeys developed recurrent hepatitis involving liver damage and cycles of HAV antigen shedding in stools. The relapses were presumably due to immune response effects.

PSA-velocity

Inclement

,Chromium – Molybdenum – Zirconium,We have studied the . gene as a possible target of HBV X protein in liver carcinogenesis. Our results indicate that .-activation by X protein occurs via PKC/AP1 signal transduction, suggesting a possible two-step mechanism in HBV related liver carcinogenesis.

GROVE

Immune pathogenesis of hepatitis AIn an effort to elucidate the mechanism of liver damage resulting from Hepatitis A virus (HAV) infection, we have studied infected skin fibroblasts and autologous lymphocytes from HAV patients. We report here that HLA-restricted virus-specific T cells play an essential role in HAV-related hepatocellular injury.

刪減

Relapsing hepatitis A in Saimiri monkeys experimentally reinfected with a wild type hepatitis A viruSaimiri monkeys were inoculated three times with hepatitis A virus and observed in a follow-up study for sixteen months. The monkeys developed recurrent hepatitis involving liver damage and cycles of HAV antigen shedding in stools. The relapses were presumably due to immune response effects.

協(xié)議

寬敞