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Titlebook: Chronic Pancreatitis; Research and Clinica H. G. Beger,M. Büchler,P. Malfertheiner Conference proceedings 1990 Springer-Verlag Berlin Heide

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樓主: aggression
21#
發(fā)表于 2025-3-25 05:22:47 | 只看該作者
Focal Necrosis: Primary Event in the Pathogenesis of Chronic Pancreatitis? and chronic pancreatitis differ in etiology and pathogenesis [18]. Here we will briefly review the pathology of focal necrosis and pseudocysts in chronic pancreatitis and discuss their possible role in the pathogenesis of the disease.
22#
發(fā)表于 2025-3-25 11:12:12 | 只看該作者
23#
發(fā)表于 2025-3-25 12:26:59 | 只看該作者
24#
發(fā)表于 2025-3-25 19:25:14 | 只看該作者
Birgit Ulrike Münch,Andreas Tackeundergo a tonsillectomy or spontaneous abortion [5], just to name a few. Chronic pancreatitis also has been mentioned among these risk factors. The purpose of this report is to focus on whether there is any link between chronic pancreatitis and pancreatic cancer based on review of the epidemiologic data and clinical and experimental experiences.
25#
發(fā)表于 2025-3-25 22:38:39 | 只看該作者
26#
發(fā)表于 2025-3-26 00:39:50 | 只看該作者
27#
發(fā)表于 2025-3-26 04:29:40 | 只看該作者
Chronic Pancreatitis with Inflammatory Mass in the Head of the Pancreas: A Special Entity?t lesion either in the right (head) or in the left part (body and tail) of the gland. For this reason, surgical techniques in chronic pancreatitis have been developed and applied either to resect the pancreatic head and preserve the body and tail or vice versa [4, 5].
28#
發(fā)表于 2025-3-26 10:07:32 | 只看該作者
Peptidergic Innervation in Chronic Pancreatitise tachykinin substance P (SP) may be involved in chronic inflammatory and painful disease of the gastrointestinal system is evidenced by a selective increase in the density of tachykinin receptors in the bowels of patients suffering from Crohn’s disease and ulcerative colitis [62, 63, 64].
29#
發(fā)表于 2025-3-26 15:38:16 | 只看該作者
30#
發(fā)表于 2025-3-26 19:25:52 | 只看該作者
Juristen um Luther in Wittenberge tachykinin substance P (SP) may be involved in chronic inflammatory and painful disease of the gastrointestinal system is evidenced by a selective increase in the density of tachykinin receptors in the bowels of patients suffering from Crohn’s disease and ulcerative colitis [62, 63, 64].
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