Titlebook: Chronic Kidney Disease and Hypertension; Matthew R. Weir,Edgar V. Lerma Book 2015 Springer Science+Business Media New York 2015 BP Monitor

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Blood Pressure Vaccines,oviders. The idea of a vaccine for hypertension that could ameliorate or cure the disease with a single procedure is innately appealing. The discovery that renin increased blood pressure raised the possibility that renin could be the key to the etiology of hypertension. Administering renin from one

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Masked Hypertension: Does It Lead to CVD or CKD?,, white-coat hypertension, while also being well described, is generally accepted as exhibiting greater risk than normotension. More recently, the identification of masked hypertension (reverse white-coat hypertension using older terminology) has added to the complexity of the hypertensive phenotype

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Uric Acid and Hypertension: Is There Really a Link?, The etiology of primary (essential) hypertension is complex, and consists of genetic, maternal–fetal, and environmental factors. Ideally, one would like to identify modifiable risk factors that could either prevent or treat hypertension. An elevated serum uric acid, which is a clearly amendable ris

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Preeclampsia: Angiogenic Factors, Blood Pressure, and the Kidney,the pathogenesis of preeclampsia (PE) have yet to be fully elucidated. PE affects the vasculature of many target maternal organs including the kidney, liver, and brain. Growing evidence supports the concept that the placenta plays a central role in the pathogenesis of PE and that reduced uteroplacen

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Inflammation and Hypertension, a condition of low-grade systemic inflammation is frequently documented in hypertensive subjects and data from animal models of hypertension demonstrated that inflammatory events are commonly seen within the target organs of hypertensive damage (such as kidneys, arteries, and the brain). Whether th

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Genome-Wide Association Studies (Gwas) of Blood Pressure in Different Populations,ed a significant heritable component to the most common form, essential hypertension. Several strategies have been developed to identify a genetic variation that is associated with hypertension. Genome-wide association studies (GWAS) were developed to identify particular genetic markers (single-nucl

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Endocrine Hypertension and Chronic Kidney Disease,tors. These conditions include primary aldosteronism and some forms of congenital adrenal hyperplasia, Cushing’s, and pheochromocytoma/paraganglioma syndromes, respectively. One needs to remember that these conditions are rare, but associate with significant morbidity and need to be included in the

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Hypertension in Children with Chronic Kidney Disease,eft ventricular hypertrophy, increased carotid intimal thickness, and some subtle neurocognitive defects. Although widely acknowledged that hypertension should be aggressively treated, the optimal target for blood pressure control in children with CKD has not been clearly defined. In most cases, pha

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