Titlebook: Cholinergic Mechanisms; Phylogenetic Aspects Giancarlo Pepeu,Herbert Ladinsky Book 1981 Springer Science+Business Media New York 1981 Foam.

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書目名稱Cholinergic Mechanisms影響因子(影響力)




書目名稱Cholinergic Mechanisms影響因子(影響力)學(xué)科排名




書目名稱Cholinergic Mechanisms網(wǎng)絡(luò)公開度




書目名稱Cholinergic Mechanisms網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cholinergic Mechanisms被引頻次




書目名稱Cholinergic Mechanisms被引頻次學(xué)科排名




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書目名稱Cholinergic Mechanisms年度引用學(xué)科排名




書目名稱Cholinergic Mechanisms讀者反饋




書目名稱Cholinergic Mechanisms讀者反饋學(xué)科排名

concubine

Questions Raised by the Electron Microscopic Localization of Acetylcholinesterase and Butyrylcholinened solely to the preganglionic fibers and their terminals. Staining for BuChE was intense throughout the neuropil and completely absent from the perikaya of the ganglion cells; following denervation, there was only a slight decrease in the intensity of staining. From these and related findings (1,2

幼稚

Somber

Parley

Kinetic Studies on the Release of [3H]-Acetylcholine from Guinea Pig Myenteric Plexus: Difference inof mechanical activity induced by stimulation. To overcome this problem, the method developed for measuring labelled ACh release from brain slices in the absence of Cholinesterase inhibitors (13) was adapted for the myenteric plexus-longitudinal muscle preparation. After labelling part of the ACh st

micronutrients

0099-6246 lation of acetylcholine synthesis, release, post- synaptic, electrical and trophic effects in the peripheral chol- inergic synapses including ganglia, neuromusc978-1-4684-8645-2978-1-4684-8643-8Series ISSN 0099-6246

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昏暗

insipid

https://doi.org/10.1007/978-3-663-13098-7tinuously modified by a number of factors, including the amount of co-transmitter that is released from the nerve terminals together with the primary transmitter (5,6) and 2) it appears that the co-existence of a primary transmitter with a given co-transmitter is not obligatory. For instance, ACh co

昏睡中

Ellen Haustein,Peter C. Lorson,Horst Zündorf (Ch) on the evoked outflow of labelled ACh in the absence of a Cholinesterase inhibitor were investigated. The measurement of ACh release from the myenteric plexus without Cholinesterase inhibition was based on the method described by Szerb (11); incubation of the myenteric plexus preparation with