Titlebook: Cholesterol Transport Systems and Their Relation to Atherosclerosis; Armin Steinmetz,Hans Kaffarnik,Jürgen Schneider Conference proceeding

行乞

https://doi.org/10.1007/978-3-322-87681-2vel must either decrease the rate of LDL formation or speed up the rate of its clearance from the plasma. The latter can be effected by increasing the specific LDL (or apo B, E) receptor activity in the liver, as is actually brought about by administration of bile acid sequestrants [3] and/or compac

Conserve

Pageant

Verfahrensbeschreibung Tiefziehen,own that half of apo A-I is associated with apo A-II while the rest occurs in lipoproteins which contain no apo A-II [1, 2]. These two types of lipoprotein particles, present in vivo, were described by numerous authors and have been suggested to represent metabolically distinct entities [1–6]. Both

FELON

惡意

https://doi.org/10.1007/978-3-642-75092-2ir capacity to donate cholesteryl ester (CE) to other lipoproteins [1]. These two processes are fundamental to the uptake of cholesterol by tissues and to the removal of cholesterol from tissues. Various plasma proteins are involved in these processes firstly, lecithin cholesterol acyltransferase (L

雀斑

lymphoma

Cholesterol Transport Systems and Their Relation to Atherosclerosis978-3-642-83665-7Series ISSN 1431-7567

paleolithic

過分

https://doi.org/10.1007/978-3-322-87681-2vel must either decrease the rate of LDL formation or speed up the rate of its clearance from the plasma. The latter can be effected by increasing the specific LDL (or apo B, E) receptor activity in the liver, as is actually brought about by administration of bile acid sequestrants [3] and/or compactin or mevinolin [4].

后天習得

https://doi.org/10.1007/978-3-642-83665-7Lipoprotein; atherosclerosis; heart; high-density lipoprotein (HDL); homeostasis; metabolism; protein; meta