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Titlebook: Cholesterol; Interactions with Te Jens M?ller Book 1987Latest edition Springer-Verlag Berlin Heidelberg 1987 biochemistry.cardiovascular.he

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發(fā)表于 2025-3-21 17:26:00 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Cholesterol
副標(biāo)題Interactions with Te
編輯Jens M?ller
視頻videohttp://file.papertrans.cn/227/226155/226155.mp4
圖書(shū)封面Titlebook: Cholesterol; Interactions with Te Jens M?ller Book 1987Latest edition Springer-Verlag Berlin Heidelberg 1987 biochemistry.cardiovascular.he
描述In 1935, Butenandt and Ruzicka received the Nobel Prize for synthesizing testosterone from cholesterol. Over the next few years, testosterone was used therapeutically by several clinicians, especially in Germany. However, with the outbreak of the Second World War, international scientific research was effectively brought to an end. Using testosterone extracted from postmortem material, workers at the clinic of the famous Danish surgeon Thorkild Rovsing were able to show as early as the First World War that the substance had beneficial effects on cardiovascular disease. Given that this particularly important chapter in medical history opened in Copenhagen, it is appropriate that testosterone treatment was taken up in the 1950s by Jens Meller. During the 35 years since then, he has gained enormous experience in treating cardiovascular disease and is amply qualified to review the role testosterone has played in the more than ten thousand patients he has treated, most of them successfully. Many have even been saved the experience of amputation. Drawing on authoritative sources and expert opinion in the fields of biochemistry and physiology, this book explains the scientific basis that
出版日期Book 1987Latest edition
關(guān)鍵詞biochemistry; cardiovascular; heart disease; physiology; vascular disease
版次1
doihttps://doi.org/10.1007/978-3-642-71600-3
isbn_softcover978-3-642-71602-7
isbn_ebook978-3-642-71600-3
copyrightSpringer-Verlag Berlin Heidelberg 1987
The information of publication is updating

書(shū)目名稱(chēng)Cholesterol影響因子(影響力)




書(shū)目名稱(chēng)Cholesterol影響因子(影響力)學(xué)科排名




書(shū)目名稱(chēng)Cholesterol網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱(chēng)Cholesterol網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱(chēng)Cholesterol被引頻次




書(shū)目名稱(chēng)Cholesterol被引頻次學(xué)科排名




書(shū)目名稱(chēng)Cholesterol年度引用




書(shū)目名稱(chēng)Cholesterol年度引用學(xué)科排名




書(shū)目名稱(chēng)Cholesterol讀者反饋




書(shū)目名稱(chēng)Cholesterol讀者反饋學(xué)科排名




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發(fā)表于 2025-3-21 23:16:47 | 只看該作者
Stress,f gluconeogenic enzymes in the liver — necessary to maintain normal glucose level. Dysstress means excess of Cortisol and catecholamines, leading to hyperglycaemia and impaired carbohydrate metabolism, i.e., CVD.
板凳
發(fā)表于 2025-3-22 02:30:20 | 只看該作者
Book 1987Latest editionsterone has played in the more than ten thousand patients he has treated, most of them successfully. Many have even been saved the experience of amputation. Drawing on authoritative sources and expert opinion in the fields of biochemistry and physiology, this book explains the scientific basis that
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發(fā)表于 2025-3-22 11:56:51 | 只看該作者
e was used therapeutically by several clinicians, especially in Germany. However, with the outbreak of the Second World War, international scientific research was effectively brought to an end. Using testosterone extracted from postmortem material, workers at the clinic of the famous Danish surgeon
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The Nature of Cardiovascular Disease, lies in the predominance of the influence of catabolism, represented by catecholamines and cortisol, over the role of anabolism, represented by testosterone and insulin, leading to an increase in cholesterol level.
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發(fā)表于 2025-3-23 02:39:52 | 只看該作者
Difficulty in Distinguishing Ageing and Pathological Processes,ite (1982, p 528), “It is often difficult to say whether degenerative changes are . in nature, but predisposition to fatal infections, malignant disease and . account for the vast majority of deaths in old people.” (Emphasis added)
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發(fā)表于 2025-3-23 06:17:07 | 只看該作者
Uwe M. Borghoff,Johann H. Schlichterized from active acetate units in lieu of their conversion to fatty acids and leads to the rapid development of arteriosclerosis.” These active acetate units are the acetyl CoA not oxidated by the Krebs cycle.
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