Titlebook: Childhood Acute Lymphoblastic Leukemia; Ajay Vora Book 2017 Springer International Publishing Switzerland 2017 acute lymphoblastic leukemi

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書目名稱Childhood Acute Lymphoblastic Leukemia影響因子(影響力)




書目名稱Childhood Acute Lymphoblastic Leukemia影響因子(影響力)學(xué)科排名




書目名稱Childhood Acute Lymphoblastic Leukemia網(wǎng)絡(luò)公開度




書目名稱Childhood Acute Lymphoblastic Leukemia網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Childhood Acute Lymphoblastic Leukemia被引頻次




書目名稱Childhood Acute Lymphoblastic Leukemia被引頻次學(xué)科排名




書目名稱Childhood Acute Lymphoblastic Leukemia年度引用




書目名稱Childhood Acute Lymphoblastic Leukemia年度引用學(xué)科排名




書目名稱Childhood Acute Lymphoblastic Leukemia讀者反饋




書目名稱Childhood Acute Lymphoblastic Leukemia讀者反饋學(xué)科排名

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https://doi.org/10.1007/978-90-481-8704-1ave a more varied presentation. Bone pain and the signs and symptoms of mediastinal and central nervous system involvement are of particular relevance. Time to presentation for medical attention is related to presenting symptoms. In this chapter, the clinical and laboratory features of ALL at presen

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HwanSuk Chris Choi,Ercan Sirakaya Turkple effector mechanisms of the antileukemic agents that are responsible for the efficacy (Fig. 5.1 and Table 5.1). This contrasts the modern era of targeted therapy, where molecular mapping of chemoresistant cancer cells has led to development of drugs that specifically target aberrant pathways (see

江湖騙子

Filippo M. Denaro,Giuliano De Stefano. MRD measurements have proven to be the strongest independent prognostic factor, allowing for risk-group assignment into different treatment arms, ranging from treatment reduction or intensification. MRD measurements are also guiding treatment decisions in relapsed ALL patients and patients undergo

江湖騙子

ERUPT

Katherine N. Marenco,David J. Bottjergic subsets of childhood ALL and improved clinical risk stratification of patients. These genomic advances have further identified key leukaemia-associated pathways and somatic alterations that may be amenable to therapeutic targeting with kinase inhibitors and other molecularly-targeted agents. Thi

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neutrophils