Titlebook: Chemical Carcinogenesis and Mutagenesis II; Colin S. Cooper,Philip L. Grover Book 1990 Springer-Verlag Berlin Heidelberg 1990 Carcinom.DNA

得罪人

AFFIX

Cancer-Prone Human Disorders with Defects in DNA Repairl as with a variety of other clinical abnormalities. The prototype disease in this category is xeroderma pigmentosum (XP) in which a clearly identifiable defect in the repair of UV damage in DNA is associated with a high frequency of UV-induced mutations in cultured cells and with a greatly increase

Pcos971

DNA Repair and Carcinogenesis by Alkylating Agents a tumor. Numerous laboratory investigations, some of which are discussed in this chapter, are consistent with this hypothesis. There is also abundant evidence from epidemiological studies of the increased incidence of cancer in individuals suffering from heritable diseases involving defects in DNA

AROMA

微塵

Inhibition of Chemical Carcinogenesisrs of chemical carcinogenesis in experimental animals. Interest in chemical inhibitors of carcinogenesis is twofold: (i) they provide valuable tools for studying the biochemical and molecular mechanism(s) of carcinogenesis; and (ii) understanding their chemical nature, mechanism(s), and specificity

包庇

ovation

刺耳的聲音

flavonoids

Signal Transduction in Proliferating Normal and Transformed Cellstion see W. et al. (1987). Following the addition of serum or defined growth factors, the quiescent cells are stimulated to enter the cell cycle. Using this experimental system, the early biochemical events occurring in the cell in response to growth stimulation can be investigated. Such studies hav

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