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Titlebook: Cerebral Energy Metabolism and Metabolic Encephalopathy; David W. McCandless Book 1985 Plenum Press, New York 1985 Nervous System.anatomy.

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書目名稱Cerebral Energy Metabolism and Metabolic Encephalopathy
編輯David W. McCandless
視頻videohttp://file.papertrans.cn/224/223300/223300.mp4
圖書封面Titlebook: Cerebral Energy Metabolism and Metabolic Encephalopathy;  David W. McCandless Book 1985 Plenum Press, New York 1985 Nervous System.anatomy.
描述In recent years, there has been rapid growth in knowledge pertaining to the nervous system. This has, in some measure, been due to the development and application of a number of techniques such as the 2-deoxyglucose method and microchemical methods for measuring metabolites and regional cerebral blood flow. Data from the application of these techniques are just beginning to be collected, and the next few years promise to bring many new and exciting findings. The study of energy metabolism in brain is particularly interesting due to the fact that although the brain has scant energy reserves (as compared with the liver), it has one of the highest metabolic rates in the body. Recent studies from several laboratories have shown a surprising divergence of re- sponses to metabolic insult in different areas of brain. In this regard, the cer- ebellum, for example, may have metabolic features which are uniqve from those of any other region. The high-energy phosphate compounds ATP and phos- phocreatine, supplied by the oxidative metabolism of glucose, are necessary for normal cerebral functions such as the maintenance of membrane potentials, transmission of impulses, and synthetic processes.
出版日期Book 1985
關(guān)鍵詞Nervous System; anatomy; behavior; cortex; metabolism; physiology; visual evoked potential (VEP)
版次1
doihttps://doi.org/10.1007/978-1-4684-1209-3
isbn_softcover978-1-4684-1211-6
isbn_ebook978-1-4684-1209-3
copyrightPlenum Press, New York 1985
The information of publication is updating

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Unterschenkel und oberes Sprunggelenkisorders, in which brain metabolism is altered secondarily to systemic changes, include hyperammonemia, hypoglycemia, nutritional deficiencies such as thiamine or niacin, some inborn errors of metabolism, and heavy metal intoxication (Plum, 1975). The metabolic encephalopathies share similar clinica
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https://doi.org/10.1007/978-3-662-09159-3Most are saturated straight-chain acids. Some are unsaturated, some branched-chain, and some hydroxylated. Fatty acids are in general hydrophobic and lipophilic. The very-short-chain fatty acids (≦ 5 carbons) are volatile and soluble in water. The medium-chain fatty acids (C.–C.) are slightly solubl
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