Titlebook: Cerebral Damage Before and After Cardiac Surgery; Allen E. Willner Book 1993 Springer Science+Business Media Dordrecht 1993 PET.Surgery.br

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Perfusion related parameters affecting cerebral outcome after cardiac surgeryopments of the 20th century. From animal laboratory experiments by physiologists more than 150 years ago to the clinical applications in the early 1950s, open-heart surgery with CPB has grown to the extent that more than 700 hospitals in the United States perform approximately 350,000 CPB procedures

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The causes of postoperative cerebral damageThe first, which follows overwhelming cerebral insult, leads to the patient’s death or severe coma. The second level concerns focal neurological deficit or ‘stroke’. This usually manifests either in hemiplegia or a retinal field defect but may selectively influence other higher cortical functions. T

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Computerized EEG in cardiac surgeryensitive to brain dysfunction [1] caused, for instance, by cerebral hypoperfusion or changing metabolism caused by different anaesthetics [2, 3]. Due to its sensitivity, the EEG provides general information concerning the condition of the patient involved. Observing the EEGs during surgery remains a

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Real time multichannel quantitative EEG monitoringmber of factors including: (1) the measure of CNS function utilized, (2) whether the study was retrospective or prospective, (3) the time span over which CNS function was monitored postoperatively, (4) the nature of the population studied, and (5) the type of cardiac procedure performed. The inciden

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Neuropsychological dysfunction before and after cardiac surgeryoperative mortality and morbidity [1]. Major neurological morbidity has also been reduced to 5% or less [2]. Whether there has been a corresponding reduction in postoperative neuropsychological impairment is still controversial. Several authors [1–8] report little postoperative neuropsychological im