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Titlebook: Central and Peripheral Mechanisms of Cardiovascular Regulation; A. Magro,W. Osswald,P. Vanhoutte Book 1986 Springer Science+Business Media

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書目名稱Central and Peripheral Mechanisms of Cardiovascular Regulation
編輯A. Magro,W. Osswald,P. Vanhoutte
視頻videohttp://file.papertrans.cn/224/223212/223212.mp4
叢書名稱NATO Science Series A:
圖書封面Titlebook: Central and Peripheral Mechanisms of Cardiovascular Regulation;  A. Magro,W. Osswald,P. Vanhoutte Book 1986 Springer Science+Business Media
描述There is increasing awareness that the autonomic nervous system, through its central and peripheral pathways, plays a critical role in the regulation of the circulation. Peripherally, the autonomic representation, largely that of sympathetic nerves, innervate virtually all segments of the vascular tree as well as the adrenal medulla. Through the interaction of nerve terminals, their transmitters, receptors and intracellular mediators in smooth muscle, sympathetic neurons control vascular tone as well as the basal performance of the heart. In turn, the performance of the autonomic nervous system is highly controlled by the brain. Once viewed as a black box with only a vague influence on cardiovascular performance, the introduction of concepts and techniques of neuroscience into the field of cardiovascular medicine has led to the realization of the critical role of this organ in cardiovascular control. It is now well recognized that within the brain, the represenation of cardiovascular function is highly restricted anatomically, engages a number of specific transmitters for its actions, and has highly selective and topographically restricted functions to influence circulatory perform
出版日期Book 1986
關(guān)鍵詞Peptid; brain; circulation; influence; medicine; nervous system; neuroscience; receptor; receptors; regulatio
版次1
doihttps://doi.org/10.1007/978-1-4615-9471-0
isbn_softcover978-1-4615-9473-4
isbn_ebook978-1-4615-9471-0
copyrightSpringer Science+Business Media New York 1986
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https://doi.org/10.1007/978-3-642-84233-7 excitation-contraction coupling. There is no doubt that the final step in the activation of smooth muscle, i.e. the force development depends on an interaction of the actin and myosin filaments. The main regulatory factor of this activity is the concentration of ionized calcium in the cytoplasm. Fr
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https://doi.org/10.1007/978-3-642-84247-4). Since that time evidence has been provided for the concept of subtypes of and ?.- and ?. -adrenoceptorss Lands et al. (1967a,b); the discovery of presynaptic α - and ?-adrenoceptors, Farnebo and Hamberger (1971); Kirpekar and Puig (1971); Langer et al. (1971); Starke (1971); Adler-Graschinsky and
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https://doi.org/10.1007/978-3-642-84286-3direct activation of the smooth muscle, to amplification of the response to other neurohumoral mediators, or to the liberation of other endogenous vasoconstrictors (e.g. norepinephrine from adrenergic nerves). Vasodilator responses to serotonin are seen mainly at the arteriolar level, but can also b
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Propagation Systems and Evolution in ,eganglionic neurons (SPNs), whose axons project to the sympathetic ganglia and the adrenal medulla, transmitting these patterns to final sites of neural or humoral integration. It is the locus of pathways descending upon SPNs and their associated interneurons from supraspinal systems, and it is the
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