Titlebook: Central Nervous System Pathology; A New Approach G. N. Kryzhanovsky Book 1986 Consultants Bureau, New York 1986 Parkinson.brain.cortex.epil

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Hans-Jürgen Pesch,Hartmut St??,Benno Kummerformation, but also its primary processing. The presence of populations of functionally different neurons with multiple connections that produce peculiarities of the structural and functional organization of the dorsal horns allow powerful GPEE to be formed in them.

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Hans-Jürgen Pesch,Hartmut St??,Benno Kummerhey are based. This is evident from the fact that the neuropathologic syndrome is a clinical expression of the pathologic system’s activity. The aim of pathogenetic therapy is to eliminate this system.

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https://doi.org/10.1007/978-1-4684-7870-9Parkinson; brain; cortex; epilepsy; nervous system; neurons; research; spinal cord

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978-1-4684-7872-3Consultants Bureau, New York 1986

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Hans-Jürgen Pesch,Hartmut St??,Benno KummerIt has been shown in Chapters 2 and 3 that the functional relationships between the nucleus neurons are disorganized and the neurons form GPEE in LGN when the inhibitory mechanisms are disturbed. A characteristic neuropathologic syndrome of photogenic epilepsy is engendered by the origin of GPEE in LGN (Kryzhanovsky et al., 1976b).

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https://doi.org/10.1007/978-3-642-78188-9The generator of pathologically enhanced excitation (GPEE) was produced in the somnogenous regulation system in order to create a model of pathologically prolonged sleep (Kryzhanovsky et al., 1978a).