Titlebook: Central Nervous System Control of the Heart; Proceedings of the I T. Stober,K. Schimrigk,D. G. Sherman Conference proceedings 1986 Martinus

Incompetent

https://doi.org/10.1007/978-3-642-70704-9e and cerebral infarction. These patients were neurologically examined on a daily basis along with 12 lead standard ECGs and long-term Holter ECGs. The brains of 14 patients who died during this study were examined for morphological alterations within the autonomic regulatory centers. Sections were

落葉劑

Monitoring beim kardialen Risikopatienten,a catecholamines in stroke patients compared to controls. In addition, most of the patients dying from stroke had myocardial lesions similar to those seen in patients with phaeochromocytoma and in animals after systemic infusion of catecholamines. Taken together, this evidence suggests altered sympa

Cloudburst

H. Sonntag,R. Larsen,H. Stephanombining the recording of the electrocerebral activity (scalp EEG in all cases; direct brain recording through stereotactically implanted electrodes in 5 patients) and the TV monitoring of the clinical manifestations..The heart rate changed significantly during the majority of seizures (61.3 %). Tac

Iatrogenic

https://doi.org/10.1007/978-1-4613-2327-3Flow; Nervous System; brainstem; cardiovascular; cardiovascular control; cardiovascular regulation; heart

專橫

978-1-4612-9430-6Martinus Nijhoff Publishing 1986

Paradox

Central Nervous System Control of the Heart978-1-4613-2327-3Series ISSN 0897-3946

使迷醉

Topics in the Neuroscienceshttp://image.papertrans.cn/c/image/223187.jpg

cushion

Anatomic Aspects of Central Nervous Cardiovascular Regulationarius (NTS). This information is then relayed to a number of brainstem and forebrain nuclei. Two important concepts emerge from connectional studies of the NTS efferent pathways. First, the NTS projects directly to some of the brainstem and hypothalamic nuclei which give rise to direct pathways that

Constituent

insipid

Rostral Solitary Tract Lesions Produce Vasopressin Dependent Hypertension in the Doge destruction of baroreceptor afferents. Neuroanatomical studies in the dog revealed that the 8 mm length of the distribution of baroreceptor afferents within the nucleus tractus solitarius makes interruption of the baroreflex pathway by lesioning this nucleus unfeasible. However, the discovery that