Titlebook: Cellular and Molecular Biology of Neuronal Development; Ira B. Black Book 1984 Plenum Press, New York 1984 Nervous System.development.neur

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978-1-4612-9686-7Plenum Press, New York 1984

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https://doi.org/10.1007/978-3-642-00781-1o derived entirely from the neural crest (proximal ganglia of nerves IX and X), while others have a more complex origin in that the epibranchial placodes contribute either to all (geniculate, petrosal, and nodose ganglia) or to some (trigeminal) of their neurons.

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https://doi.org/10.1007/978-3-642-00820-7the migration and compartmentalization of young neurons and, later, in their maintenance and the regulation of their environment. Furthermore, it appears that genetically or environmentally induced disturbances of neuronal—glial interactions during development may lead to structural, biochemical, an

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https://doi.org/10.1007/978-3-642-01467-3n is that certain rules that govern synapse formation can be best understood by observing the behavior of individual cells in a controlled environment over periods of time that range from seconds to days. This observation is difficult if not impossible to achieve in intact embryos. The ability to vi

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,Software zur L?sung und Modellierung,ack and Patterson, 1980; Le Douarin, 1980; Black, 1982). While autonomic neurons, which have been examined in particular detail, may alter a number of transmitter characters, and even acquire new transmitters in culture (for reviews, see Patterson, 1978; Varon and Bunge, 1978), relevance to . events