Titlebook: Cellular and Molecular Basis of Synaptic Transmission; Herbert Zimmermann Conference proceedings 1988 Springer-Verlag Berlin Heidelberg 19

薄荷醇

Ocular Adnexal Lymphoproliferative Disease,n seems fairly uniform, as regards the axon terminals and the complexes formed by pre- and postsynaptic membranes, and by the electron-dense structures attached to the cytoplasmic surfaces of these membranes. However, the same does not always apply to the deep part of the subsynaptic cytoplasm, whic

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Modern Concepts in Orbital Imaging,to cause the reduction of synaptic vesicles (SVs) in number and simultaneously to depress the postsynaptic response (Ceccarelli et al., 1973; Heuser and Reese, 1973; Pysh and Wiley, 1974; Zimmerman and Whittaker, 1974a, b). Vesicular release of transmitter via exocytosis is supposed to be followed b

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https://doi.org/10.1007/978-3-540-85673-34, Zimmermann, 1982). A calcium mediated mechanism fuses vesicles with the presynaptic membrane resulting in the release of transmitter into the synaptic cleft. The increase in internal calcium results from an inward calcium current through a voltage dependent calcium channel which is activated by t