Titlebook: Cellular Peptidases in Immune Functions and Diseases 2; Jürgen Langner,Siegfried Ansorge Book 2002 The Editor(s) (if applicable) and The A

SPARK

書目名稱Cellular Peptidases in Immune Functions and Diseases 2影響因子(影響力)




書目名稱Cellular Peptidases in Immune Functions and Diseases 2影響因子(影響力)學(xué)科排名




書目名稱Cellular Peptidases in Immune Functions and Diseases 2網(wǎng)絡(luò)公開度




書目名稱Cellular Peptidases in Immune Functions and Diseases 2網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cellular Peptidases in Immune Functions and Diseases 2被引頻次




書目名稱Cellular Peptidases in Immune Functions and Diseases 2被引頻次學(xué)科排名




書目名稱Cellular Peptidases in Immune Functions and Diseases 2年度引用




書目名稱Cellular Peptidases in Immune Functions and Diseases 2年度引用學(xué)科排名




書目名稱Cellular Peptidases in Immune Functions and Diseases 2讀者反饋




書目名稱Cellular Peptidases in Immune Functions and Diseases 2讀者反饋學(xué)科排名

描述

Unterschenkel und oberes Sprunggelenk,nce of appropriate protease inhibitors..Clinical implications of peptide processing by DPP IV include that the therapeutic potential of peptides that are degraded by DPP IV may largely be enhanced by creating DPP IV-resistant, active analogues, and that the most suitable NH2-terminal modification ma

LUCY

Unterschenkel und oberes Sprunggelenk,-induced signalling pathways, including Ca2+ mobilisation, MEK1-, Erk1/2-and PKB-activation, can be strongly affected by DP IV inhibition. Thus, the enzymatic activity or at least the interaction of effectors with the catalytic domain of CD26 seems to be important for crucial functions of this cell

Airtight

Review: The Role of Membrane Peptidase in Immune Functions

COST

Coronary-Spasm

Cell-Cell Contact Between Lymphocytes and Fibroblast-Like Synovioctyes Induces Lymphocytic Expressio

Coronary-Spasm

ensemble

Transforming Growth Factor-β Increases the Expression of Aminopeptidase N/CD13 mRNA and Protein in M In experiments with actinomycin D- treated cells was found a stabilization of APN/CD13 mRNA by TGF-β1. Contrary to the IL-4-induc edexpression of APN/CD13 as well as of MHC class II in monocytic cells, we could show that TGF-β is able to augment the APN/CD13 expression but decreases the MHC class I

墻壁

Natural Substrates of Dipeptidyl Peptidase IVnce of appropriate protease inhibitors..Clinical implications of peptide processing by DPP IV include that the therapeutic potential of peptides that are degraded by DPP IV may largely be enhanced by creating DPP IV-resistant, active analogues, and that the most suitable NH2-terminal modification ma

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