Titlebook: Cellular Metabolism of the Arterial Wall and Central Nervous System; Selected Aspects Gotthard Schettler (Leiter der Geomedizinischen Fo Co

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Receptor Mediated Hepatic Uptake of Lipoproteins,ontaining lipoproteins such as low density lipoproteins (LDL) and their partially catabolized precursors, very low density lipoprotein (VLDL) remnants, from the blood (1,2). Yet, the removal mechanisms for chylomicron remnants and high density lipoproteins (HDL) have still to be solved.

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Common Mutations Underlying the Hypertriglyceridaemia/Low HDL Syndrome,hen the fasting plasma triglycérides are greater than 2.2 mmol/1 and HDL-cholesterol less than 1.0 mmol/1. This dyslipidaemia is usually classified into primary (implying genetic cases) or secondary types to other disorders such as diabetes mellitus, obesity, and alcohol excess. However in the commo

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Studies on Lipoprotein Lipase,n plateaus [1, 2]. Expression of LPL requires the presence of 20% serum and activity decreases markedly in serum-poor medium [3,4], Basic fibroblast growth factor (bFGF) plays an important role in cell proliferation and presently we have inquired whether it may replace serum in our system and whethe

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Biochemical and Molecular Characterization of the Scavenger Receptors,riments to learn by what mechanisms the arterial wall accumulates cholesterol from plasma low density lipoprotein (LDL) in patients with Familial Hypercholesterolemia (FH) suffering from premature atherosclerosis. The plasma LDL levels of FH patients are dramatically elevated and atherosclerosis beg

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Conference proceedings 1993r biology of atherogenesis, plasma lipproteins, lipid transport, long chain fatty acid transport and metabolism, and lipoprotein receptors. Future directions and perspectives and common features in these important areas are discussed.