Titlebook: Cellular Interactions in Cardiac Pathophysiology; Ján Slezák,Attila Ziegelh?ffer Book 1995 Springer Science+Business Media Dordrecht 1995

Expostulate

Christiane Schiersmann,Heinz-Ulrich Thielmples were obtained during surgery from patients with normoxemic (ventricular and atrial septal defects) and hypoxemic (tetralogy of Fallot) diseases. Protein fractions were isolated by stepwise extraction from both right ventricular and atrial musculature. The concentration of total atrial protein

Confound

細(xì)胞

https://doi.org/10.1007/978-3-531-92257-7 and changes in cardiac metabolism or heart rate elicited by modification of thyroid hormone levels. Capillary growth in all these conditions can be linked with increased coronary blood flow, decreased heart rate, or both. This paper brings evidence that, although increased blood flow due to long-te

Ferritin

https://doi.org/10.1007/978-3-531-92285-0the human coronary artery in which the remarkable wall thickness is due namely by the intima thickness. Deformation in length and diameter of conduit coronary arteries, due to the left and right ventricle volume increase, has been defined in non-beating canine heart. Ramus interventricularis anterio

失望未來(lái)

Beratung im betrieblichen Bildungsmanagementure subsequent to hypertrophy remains a major clinical problem. Recently, oxidative stress has been suggested to play a critical role in the pathogenesis of heart failure. Here we describe antioxidant changes as well as their significance during hypertrophy and heart failure stages. Heart hypertroph