Titlebook: Cell and Molecular Biology of Endometrial Carcinoma; Hiroyuki Kuramoto (Professor),Masato Nishida (Pres Book 2003 Springer Japan 2003 angi

暖昧關(guān)系

HEC-1 Cells: Establishment of an In Vitro Experimental System in Endometrial Carcinomaum and endometrial carcinoma at the level of a simplified cellular system; contributing to cell and molecular biological studies on endometrial carcinoma. Once a cell line is established, it provides a stable experimental system that facilitates and progresses the study of the tissues and/or neoplas

Capitulate

勉勵(lì)

Endometrial Stress Neuropeptides: Paracrine Effects on Cell Proliferation and Apoptosissteroids. Indeed, the two major types of human endometrial cells, namely epithelial and stromal, produce, among other neuropeptides, the stress-associated peptides corticotropin-releasing hormone (CRH), endorphins, and dynorphins. Multiple lines of evidence suggest that the stress-associated neurope

COM

ironic

Involvement of Cell Cycle Regulators in Steroid Hormone-Induced Growth of Endometrial Carcinomandular cells and estrogen receptor (ER)/pregesterone receptor (PR)-positive endometrial carcinoma Ishikawa cells. The results indicated that the estradiol (E2)-induced upregulation of cydin Dl may play a crucial role in the growth of normal endometrial glandular cells and is mediated by c-Jun via an

一再煩擾

conceal

Estrogen Receptor Signaling and Crosstalk with the Ah Receptor in Endometrial Cancer Cellsirth, indicating that “l(fā)ifetime” exposure to estrogens increases the incidence of both tumors. In contrast, smoking protects against the development of endometrial cancer, whereas the role of smoking in breast cancer incidence is equivocal and may be dependent on the timing and duration of smoking.

Anthem

切掉

Inhibition of AKT Oncogenic Pathway in Endometrial Cancer Cellsctivation of AKT in a panel of endometrial cancer cell lines and tumor samples. Two endometrial cancer cell lines, namely Ishikawa and RL-95 cells, and several tumor samples showed elevated levels of phosphorylated AKT. The expression of PTEN significantly suppressed the growth of Ishikawa cancer ce

減去

A New Paradigm for Vascular Endothelial Cell Growth Factor Receptor Signaling in Adult Endotheliumrocess that is dependent on the growth of new blood vessels. Outside of the female reproductive tract, the majority of the adult vasculature is quiescent and angiogenesis occurs only as part of the body’s repair processes or in diseases including diabetic retinopathy, rheumatoid arthritis, atheroscl