Titlebook: Cell Migration in Inflammation and Immunity; Methods and Protocol Daniele D’Ambrosio,Francesco Sinigaglia Book 2003 Humana Press 2003

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Interaction of Viral Chemokine Inhibitors with Chemokines,ese mechanisms have been identified in large DNA viruses such as herpesviruses and poxviruses. The exceptions are the chemokinelike activity of the human immunodeficiency virus (HIV) Tat protein and the mimicry of fractalkine by the glycoprotein G of respiratory syncytial virus.

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Animal Models to Study Chemokine Receptor Function In Vivo,ic lung disease have been used to dissect the complex pathophysiological mechanisms underlying the asthma phenotype. This increase in usage of mouse models stems from the range of tools available for outlining functional pathways for individual cells and mediators. Genetic technologies allow us to m

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Assessing the Role of Multiple Phosphoinositide 3-Kinases in Chemokine Signaling,imulated cell migration. PI3Ks can be divided into three main classes on the basis of their in vitro lipid substrate specificity, structure, and likely mode of regulation (.). Hence, the class I PI3Ks can phosphorylate PI, PI(4)P, and PI(4,5).. to yield the 3′-phosphorylated phosphoinositide (PI) li

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Real-Time In Vitro Assay for Studying Chemoattractant-Triggered Leukocyte Transendothelial Migratior endothelial counterligands (.–.). Following rolling and arrest on the endothelium, circulating immune cells locomote on and extravasate through the endothelial cell (EC) barrier (.–.). These steps must be delicately coordinated to allow leukocyte motility while maintaining leukocyte resistance to

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https://doi.org/10.1007/b137137kocytes in tissues under physiological and pathological conditions are orderly (typically neutrophils precede mononuclear cells) and selective because, in certain states, one or more leukocyte subsets are recruited preferentially (e.g., eosinophils in allergy). The current paradigm of recruitment is