Titlebook: Cell Engineering; Apoptosis Mohamed Al-Rubeai,Martin Fussenegger Book 2004 Springer Science+Business Media B.V. 2004 Mammalia.Regulation.Ti

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書目名稱Cell Engineering影響因子(影響力)




書目名稱Cell Engineering影響因子(影響力)學(xué)科排名




書目名稱Cell Engineering網(wǎng)絡(luò)公開度




書目名稱Cell Engineering網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cell Engineering被引頻次




書目名稱Cell Engineering被引頻次學(xué)科排名




書目名稱Cell Engineering年度引用




書目名稱Cell Engineering年度引用學(xué)科排名




書目名稱Cell Engineering讀者反饋




書目名稱Cell Engineering讀者反饋學(xué)科排名

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978-90-481-6615-2Springer Science+Business Media B.V. 2004

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https://doi.org/10.1007/978-981-16-5613-2 chemical basis of these interactions is a major key to understanding the mechanisms of this diverse pathway, while controlling the outcome of these interactions offers remarkable benefits to the treatment of many pathological diseases. It is now undisputed that the Bcl-2 proteins regulate the mitoc

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Teruhisa Tsuzuki,Tamie NakajimaF signalling mechanisms contribute to cell transformation, tumour progression and metastasis. Many questions remain to be answered as to how exactly the IGF system mediates its effects both in normal and tumour cells and how the IGF-1R interacting proteins and downstream signalling cascades are regu

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Tamie Nakajima,Keiko Nakamura,Akihiko Kondohvel pathways and regulators will be elucidated and new targets for intervention will become available. As discussed in this review, work in many laboratories has indicated that manipulating the apoptosis machinery can, indeed, result in suppression of apoptosis and an extension of the productive lif

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