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Titlebook: Cell Cycle and Oncogenes; 10.-12. April 1986 Widmar Tanner,Dieter Gallwitz Conference proceedings 1986 Springer-Verlag Berlin Heidelberg 19

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發(fā)表于 2025-3-21 18:43:49 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Cell Cycle and Oncogenes
副標(biāo)題10.-12. April 1986
編輯Widmar Tanner,Dieter Gallwitz
視頻videohttp://file.papertrans.cn/223/222797/222797.mp4
叢書名稱Colloquium der Gesellschaft für Biologische Chemie in Mosbach Baden
圖書封面Titlebook: Cell Cycle and Oncogenes; 10.-12. April 1986 Widmar Tanner,Dieter Gallwitz Conference proceedings 1986 Springer-Verlag Berlin Heidelberg 19
出版日期Conference proceedings 1986
關(guān)鍵詞cell; cell cycle; oncogene; oncogenes
版次1
doihttps://doi.org/10.1007/978-3-642-71686-7
isbn_softcover978-3-642-71688-1
isbn_ebook978-3-642-71686-7Series ISSN 0366-5887
issn_series 0366-5887
copyrightSpringer-Verlag Berlin Heidelberg 1986
The information of publication is updating

書目名稱Cell Cycle and Oncogenes影響因子(影響力)




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沙發(fā)
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Biochemical Properties of Oncogene-Coded Proteinsde of signal transmission from the membrane-associated tyrosine kinase through putative second messengers to the nucleus which results in proliferation, is largely unknown. Phosphatidyl-inositol and protein kinase C may be indirectly involved (Moelling et al. 1984, Sugano and Hanafusa 1985). The sec
地板
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https://doi.org/10.1007/978-981-10-4813-5ly role for c-. in inducing the “competent state” in fibroblasts and thus in the movement of quiescent cells into the cell cycle. In addition, we will discuss the signal transduction pathways that seem to be involved in the induction of c-. by growth factors.
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https://doi.org/10.1007/978-981-10-5598-0ther at the beginning of the first exon. However, two proteins of 64 and 67 K, located in the nucleus were shown to be coded for by the c-. gene. Their function is not yet understood (Hann and Eisenman 1984, Persson et al. 1984, Persson and Leder 1984).
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發(fā)表于 2025-3-23 04:55:56 | 只看該作者
Structure and Function of , and , Oncogenesficient for oncogenic activation. Furthermore, there is as yet no unequivocal identification of the relevant biochemical function(s) of the protein products of both the mutant (transduced) and the normal (chromosomal) allele of an oncogene. Hence, the molecular mechanisms of cell transformation by v-. genes are still obscure.
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