Titlebook: Cell Cycle Deregulation in Cancer; Greg H. Enders Book 2010 Springer Science+Business Media, LLC 2010 DNA.biology.cancer.cancer therapy.ce

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書目名稱Cell Cycle Deregulation in Cancer影響因子(影響力)




書目名稱Cell Cycle Deregulation in Cancer影響因子(影響力)學(xué)科排名




書目名稱Cell Cycle Deregulation in Cancer網(wǎng)絡(luò)公開度




書目名稱Cell Cycle Deregulation in Cancer網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cell Cycle Deregulation in Cancer被引頻次




書目名稱Cell Cycle Deregulation in Cancer被引頻次學(xué)科排名




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書目名稱Cell Cycle Deregulation in Cancer年度引用學(xué)科排名




書目名稱Cell Cycle Deregulation in Cancer讀者反饋




書目名稱Cell Cycle Deregulation in Cancer讀者反饋學(xué)科排名

EVICT

https://doi.org/10.1007/978-94-017-6766-8feration is a fundamental feature of all types of cancer. One of the key regulators of cell proliferation is the E2F transcription factor. E2F controls the expression of many genes that are required for cells to divide and elevated E2F activity is found in most tumor cells. The activation and inacti

Criteria

https://doi.org/10.1007/978-94-017-6766-8tiation of DNA replication, and virtually all components of the pre-RC are regulated by complex mechanisms that center around the activity of cyclin-dependent kinases (CDKs). CDK/cyclin complexes both positively and negatively regulate pre-RC components, including their expression, activity, stabili

Legion

依法逮捕

inflame

The Essence of Acts of Empathy,apoptosis. Recently, both of these proteins were found to be intimately tied to metabolic pathways and to play surprising roles in autophagy. Autophagy (“self-eating”) is a critical response of eukaryotic cells to stress. During this process, portions of the cytosol, including cytoplasmic organelles

inflame

https://doi.org/10.1007/978-94-017-7402-4 the biological organization of tumours. These cells, named tumour-initiating cells (TICs), or cancer stem cells (CSCs), share phenotypic and functional characteristics with normal stem cells, most notably their ability to self-renew. Here we discuss regulation of self-renewal in normal haematopoiet

transdermal

Canyon

ANTI

https://doi.org/10.1007/978-94-017-9984-3rogression to fully fledged cancer would allow for chemoprevention or therapeutic interventions; however in most cases, IEN remains undetected and the exact mechanisms for progression remain unknown. Barrett’s oesophagus, the premalignant stage of oesophageal adenocarcinoma, is the perfect model to