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Titlebook: Cell Biology of Herpes Viruses; Klaus Osterrieder Book 2017 The Editor(s) (if applicable) and The Author(s), under exclusive license to Sp

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發(fā)表于 2025-3-21 16:18:08 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Cell Biology of Herpes Viruses
編輯Klaus Osterrieder
視頻videohttp://file.papertrans.cn/223/222760/222760.mp4
概述Highlights the general principles of herpesvirus infection with equal attention to overall principle and important difference.Covers the cell-type- and life-style dependent differences in the establis
叢書(shū)名稱Advances in Anatomy, Embryology and Cell Biology
圖書(shū)封面Titlebook: Cell Biology of Herpes Viruses;  Klaus Osterrieder Book 2017 The Editor(s) (if applicable) and The Author(s), under exclusive license to Sp
描述Herpes viruses are widely distributed in nature, causing disease in organisms as diverse as bivalves and primates, including humans. Each virus appears to have established a long-standing relationship with its host, and the viruses have the ability to manipulate and control the metabolism of host cells, as well as innate and adaptive antiviral immune responses. Herpes viruses maintain themselves within hosts in a latent state resulting in virus persistence for years – usually for the life span of the hosts. Herpes viruses comprise a large number of pathogens with diverse cellular targets and biological consequences of infection. What they have in common is their structure and the fact that they establish a dormant (latent) infection in their hosts that usually persists for life. The reviews here will highlight the general principles of herpes virus infection, with equal attention to overall principle and important difference. Also, the cell type- and life-style dependent differences inthe establishment and maintenance of virus persistence will be covered.
出版日期Book 2017
關(guān)鍵詞innate immunity; virus release; virus replication; virus entry; virus assembly
版次1
doihttps://doi.org/10.1007/978-3-319-53168-7
isbn_softcover978-3-319-53167-0
isbn_ebook978-3-319-53168-7Series ISSN 0301-5556 Series E-ISSN 2192-7065
issn_series 0301-5556
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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The Pathogenesis of Osteoporosis,cephalitic infection. During productive lytic infection, over 80 viral proteins are expressed in a highly regulated manner, resulting in the replication of viral genomes and assembly of progeny virions. The virion of all herpesviruses consists of an external membrane envelope, a proteinaceous layer
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Background to disease of the earThis requires passage of the genome-containing capsid across the double-membrane nuclear envelope. Herpesviruses have evolved a mechanism that relies on a pair of conserved viral gene products to shuttle the capsids from the nucleus to the cytoplasm by way of envelopment and de-envelopment at the in
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