Titlebook: Cardiovascular Pharmacology of 5-Hydroxytryptamine; Prospective Therapeu P. R. Saxena,D. I. Wallis,P. Bevan Book 1990 Springer Science+Busi

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0166-9842 Overview: 978-94-010-6701-0978-94-009-0479-8Series ISSN 0166-9842

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Benjamin Fine,Gerhard Rosenbergernstriction or in autonomic neurones which alter cardiac output. In addition, cardiovascular responses will be modified if 5-HT modulates the output of transmitter at the neuroeffector junction. Cardiovascular actions may also result from 5-HT affecting the afferent limb of reflex pathways.

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5-Hydroxytryptamine and related drugs and autonomic ganglianstriction or in autonomic neurones which alter cardiac output. In addition, cardiovascular responses will be modified if 5-HT modulates the output of transmitter at the neuroeffector junction. Cardiovascular actions may also result from 5-HT affecting the afferent limb of reflex pathways.

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The distribution and biochemistry of 5-hydroxytryptamine in the cardiovascular systemthe pineal gland and the platelets contain important concentrations of 5-HT. The indoleamine has also been detected in peripheral nerves of the gut, in lung, kidney, spleen, thyroid, mast cells, heart and blood vessels of different species. It is well accepted that 5-HT has a role as a neurotransmit

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Cloning of the choroid plexus 5-HT1C receptor homologies among one group of receptors, the G protein-coupled receptors, that unite these proteins into one structural family [1]. Members of this family include the muscarinic cholinoceptors, alpha- and beta-adrenoceptors, opsin and the Substance K receptor [1, 2]. All members of this family are

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