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Titlebook: Cardiovascular Genomics: New Pathophysiological Concepts; Proceedings of the 2 Pieter A. Doevendans,Stefan K??b Conference proceedings 2002

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書(shū)目名稱(chēng)Cardiovascular Genomics: New Pathophysiological Concepts
副標(biāo)題Proceedings of the 2
編輯Pieter A. Doevendans,Stefan K??b
視頻videohttp://file.papertrans.cn/222/221946/221946.mp4
叢書(shū)名稱(chēng)Developments in Cardiovascular Medicine
圖書(shū)封面Titlebook: Cardiovascular Genomics: New Pathophysiological Concepts; Proceedings of the 2 Pieter A. Doevendans,Stefan K??b Conference proceedings 2002
描述Four years ago-in December 1997-the first European Science Foundation Workshop on Cardiovascular Specific Gene Expression was held in Maastricht. It was hardly possible to imagine the progress in the field in those four years. In 1997, gene expression was still an art focused on individual genes; in 200 I, many labs have access to micro-array facilities to determine the expression of thousands of genes simultaneously. In 1997, gene expression was an area of fundamental research in basis molecular biology laboratories; in 200 I, clinical cardiovascular research has incorporated gene expression approaches. In 1997, the interpretation of a gene expression experiment was usually straightforward; in 200 I, advanced bioinformatics tools are needed to approach the extreme complexities of genetic control of cell and tissue function. The second symposium in this series is focused on Cardiovascular Genomics. New Pathophysiological Concepts. The organizing committee chose to invite a group of renown scientists and young investigators around four topics of eminent importance in cardiovascular research. These topics reflect the major present-day clinical cardiovascular problems: atherosclerosis
出版日期Conference proceedings 2002
關(guān)鍵詞Microarray; cardiovascular; gene expression; gene therapy; genes; genetics; hypertension; molecular mechani
版次1
doihttps://doi.org/10.1007/978-1-4615-1005-5
isbn_softcover978-1-4613-5347-8
isbn_ebook978-1-4615-1005-5Series ISSN 0166-9842
issn_series 0166-9842
copyrightSpringer Science+Business Media New York 2002
The information of publication is updating

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1.8 Glossary of general abbreviations,r than single culprit genes form the basis for the widespread penetrance of the disease in the elderly Western population. We are applying functional Genomics to the study of atherosclerosis, with the goal of characterizing healthy and diseased gene expression profiles. While our immediate objective
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53-98 - 53-161: (C4H10HgI2O2)x - C6H5IO,ure and prevention or reversal of end-organ damage with few side effects. This work will review current vascular gene transfer for hypertension with particular emphasis on.gene transfer in models of essential hypertension.
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52-220 - 52-261: F6O4SSb2.2H4N - Sb2Se3, these modifier genes have been recalcitrant to direct identification in human populations. In this regard, genetic studies in animals models of disease can identify candidate modifier genes and provide an insight on the genetic interactions that cause phenotypic variation in the human population[..
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1.8 Glossary of general abbreviations,oss of myocardial contractile function. The degeneration of structure and function in cardiomyocytes observed in dilation is less amenable to.analysis compared to hypertrophie changes associated with enhanced contractile function and enlarged cardiomyocyte size that are readily apparent by analyses
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